Publications (68)

2020

  1. Mitochondrial base excision repair positively correlates with longevity in the liver and heart of mammals

    GeroScience, Vol. 42, Núm. 2, pp. 653-665

2016

  1. Rapamycin reverses age-related increases in mitochondrial ROS production at complex I, oxidative stress, accumulation of mtDNA fragments inside nuclear DNA, and lipofuscin level, and increases autophagy, in the liver of middle-aged mice

    Experimental Gerontology, Vol. 83, pp. 130-138

2014

  1. Lifelong treatment with atenolol decreases membrane fatty acid unsaturation and oxidative stress in heart and skeletal muscle mitochondria and improves immunity and behavior, without changing mice longevity

    Aging Cell, Vol. 13, Núm. 3, pp. 551-560

  2. Independent and additive effects of atenolol and methionine restriction on lowering rat heart mitochondria oxidative stress

    Journal of Bioenergetics and Biomembranes, Vol. 46, Núm. 3, pp. 159-172

2012

  1. Effects of aging and methionine restriction applied at old age on ROS generation and oxidative damage in rat liver mitochondria

    Biogerontology, Vol. 13, Núm. 4, pp. 399-411

2011

  1. Methionine and homocysteine modulate the rate of ROS generation of isolated mitochondria in vitro

    Journal of Bioenergetics and Biomembranes, Vol. 43, Núm. 4, pp. 377-386

  2. Forty percent methionine restriction lowers DNAmethylation, complex i ROS generation, and oxidative damage to mtDNA and mitochondrial proteins in rat heart

    Journal of Bioenergetics and Biomembranes, Vol. 43, Núm. 6, pp. 699-708

2010

  1. The β-blocker atenolol lowers the longevity-related degree of fatty acid unsaturation, decreases protein oxidative damage, and increases extracellular signal-regulated kinase signaling in the heart of C57BL/6 mice

    Rejuvenation Research, Vol. 13, Núm. 6, pp. 683-693

2009

  1. Forty percent methionine restriction decreases mitochondrial oxygen radical production and leak at complex I during forward electron flow and lowers oxidative damage to proteins and mitochondrial DNA in rat kidney and brain mitochondria.

    Rejuvenation research, Vol. 12, Núm. 6, pp. 421-434

  2. Effect of methionine dietary supplementation on mitochondrial oxygen radical generation and oxidative DNA damage in rat liver and heart

    Journal of Bioenergetics and Biomembranes, Vol. 41, Núm. 3, pp. 309-321

  3. Effect of 40% restriction of dietary amino acids (except methionine) on mitochondrial oxidative stress and biogenesis, AIF and SIRT1 in rat liver

    Biogerontology, Vol. 10, Núm. 5, pp. 579-592

  4. Efecto de la restricción de aminoácidos excepto la metionina sobre el estrés oxidativo mitocondrial

    Revista Espanola de Geriatria y Gerontologia, Vol. 44, Núm. 4, pp. 194-199

2008

  1. Restricción calórica, estrés oxidativo y longevidad

    Revista Espanola de Geriatria y Gerontologia, Vol. 43, Núm. 4, pp. 252-260

  2. Lowered methionine ingestion as responsible for the decrease in rodent mitochondrial oxidative stress in protein and dietary restriction. Possible implications for humans

    Biochimica et Biophysica Acta - General Subjects, Vol. 1780, Núm. 11, pp. 1337-1347

  3. Forty percent and eighty percent methionine restriction decrease mitochondrial ROS generation and oxidative stress in rat liver

    Biogerontology, Vol. 9, Núm. 3, pp. 183-196

  4. Effect of every other day feeding on mitochondrial free radical production and oxidative stress in mouse liver

    Rejuvenation Research, Vol. 11, Núm. 3, pp. 621-629

2006

  1. La radio por Internet: la radio sin fronteras

    Razón y palabra, Núm. 49, pp. 53

2002

  1. Influence of aging and long-term caloric restriction on oxygen radical generation and oxidative DNA damage in rat liver mitochondria

    Free Radical Biology and Medicine, Vol. 32, Núm. 9, pp. 882-889

  2. Effect of time of restriction on the decrease in mitochondrial H2O2 production and oxidative DNA damage in the heart of food-restricted rats

    Microscopy Research and Technique, Vol. 59, Núm. 4, pp. 273-277

2001

  1. Caloric restriction decreases mitochondrial free radical generation at complex I and lowers oxidative damage to mitochondrial DNA in the rat heart.

    The FASEB journal : official publication of the Federation of American Societies for Experimental Biology, Vol. 15, Núm. 9, pp. 1589-1591