Publicaciones en las que colabora con Alba Naudí (27)

2022

  1. Elovl2-Ablation Leads to Mitochondrial Membrane Fatty Acid Remodeling and Reduced Efficiency in Mouse Liver Mitochondria

    Nutrients, Vol. 14, Núm. 3

2020

  1. Gene expression and regulatory factors of the mechanistic target of rapamycin (mTOR) complex 1 predict mammalian longevity

    GeroScience, Vol. 42, Núm. 4, pp. 1157-1173

  2. Low abundance of NDUFV2 and NDUFS4 subunits of the hydrophilic complex I domain and VDAC1 predicts mammalian longevity

    Redox Biology, Vol. 34

2016

  1. Human Aging Is a Metabolome-related Matter of Gender

    Journals of Gerontology - Series A Biological Sciences and Medical Sciences, Vol. 71, Núm. 5, pp. 578-585

  2. Rapamycin reverses age-related increases in mitochondrial ROS production at complex I, oxidative stress, accumulation of mtDNA fragments inside nuclear DNA, and lipofuscin level, and increases autophagy, in the liver of middle-aged mice

    Experimental Gerontology, Vol. 83, pp. 130-138

2015

  1. Cysteine dietary supplementation reverses the decrease in mitochondrial ROS production at complex I induced by methionine restriction

    Journal of Bioenergetics and Biomembranes, Vol. 47, Núm. 3, pp. 199-208

  2. Long lifespans have evolved with long and monounsaturated fatty acids in birds

    Evolution, Vol. 69, Núm. 10, pp. 2776-2784

2014

  1. Independent and additive effects of atenolol and methionine restriction on lowering rat heart mitochondria oxidative stress

    Journal of Bioenergetics and Biomembranes, Vol. 46, Núm. 3, pp. 159-172

  2. Lifelong treatment with atenolol decreases membrane fatty acid unsaturation and oxidative stress in heart and skeletal muscle mitochondria and improves immunity and behavior, without changing mice longevity

    Aging Cell, Vol. 13, Núm. 3, pp. 551-560

2013

  1. Exceptionally old mice are highly resistant to lipoxidation-derived molecular damage

    Age, Vol. 35, Núm. 3, pp. 621-635

  2. Formation of S-(carboxymethyl)-cysteine in rat liver mitochondrial proteins: Effects of caloric and methionine restriction

    Amino Acids, Vol. 44, Núm. 2, pp. 361-371

  3. La suplementación de larga duración con atenolol convierte la insaturación de las membranas mitocondriales de músculo esquelético y de corazón de ratón en la característica de mamíferos de supervivencia diez veces mayor. Efectos sobre la longevidad media y máxima

    Anales de la Real Academia Nacional de Farmacia, Vol. 79, Núm. 2, pp. 253-273

  4. Membrane lipid unsaturation as physiological adaptation to animal longevity

    Frontiers in Physiology, Vol. 4 DEC

  5. Plasma long-chain free fatty acids predict mammalian longevity

    Scientific Reports, Vol. 3

  6. Reduction in mitochondrialmembrane peroxidizability index and protein lipoxidation levels in therat heart after ß-adrenergic receptor signaling interruption with the ß-blocker atenolol

    Anales de la Real Academia Nacional de Farmacia, Núm. 4, pp. 613-633

2012

  1. Effects of aging and methionine restriction applied at old age on ROS generation and oxidative damage in rat liver mitochondria

    Biogerontology, Vol. 13, Núm. 4, pp. 399-411

2011

  1. Forty percent methionine restriction lowers DNAmethylation, complex i ROS generation, and oxidative damage to mtDNA and mitochondrial proteins in rat heart

    Journal of Bioenergetics and Biomembranes, Vol. 43, Núm. 6, pp. 699-708

2010

  1. The β-blocker atenolol lowers the longevity-related degree of fatty acid unsaturation, decreases protein oxidative damage, and increases extracellular signal-regulated kinase signaling in the heart of C57BL/6 mice

    Rejuvenation Research, Vol. 13, Núm. 6, pp. 683-693

2009

  1. Bases biológicas del proceso de envejecimiento y la longevidad

    Actualizaciones en aspectos básicos y clínicos del envejecimiento y la fragilidad (Sociedad Española de Medicina Geriátrica), pp. 21-42

  2. Effect of 40% restriction of dietary amino acids (except methionine) on mitochondrial oxidative stress and biogenesis, AIF and SIRT1 in rat liver

    Biogerontology, Vol. 10, Núm. 5, pp. 579-592