Marcadores snp asociados a terneza en dos músculos, Flexor Digitorum y Psoas Major, en avileña negra ibérica

  1. Ximena Quintero Arboleda 1
  2. MªJesús Carabaño 1
  3. Guillherme Venturini 2
  4. Cristina Meneses 1
  5. Julia Rueda 3
  6. Clara Díaz 1
  1. 1 INIA
  2. 2 Universidade Estadual Paulista
    info

    Universidade Estadual Paulista

    São Paulo, Brasil

    ROR https://ror.org/00987cb86

  3. 3 Universidad Complutense de Madrid
    info

    Universidad Complutense de Madrid

    Madrid, España

    ROR 02p0gd045

Book:
XV Jornadas sobre Producción Animal: 14 y 15 de mayo de 2013, Zaragoza
  1. Jorge Hugo Calvo Lacosta
  2. Isabel Casasús Pueyo
  3. Margalida Joy Torrens
  4. Javier Álvarez Rodríguez
  5. Luis Varona Aguado
  6. Begoña Panea Doblao
  7. Carlos Calvete Margolles
  8. Joaquim Balcells Teres

Publisher: Asociación Interprofesional para el Desarrollo Agrario

ISBN: 978-84-695-7684-7 978-84-695-7684-7

Year of publication: 2013

Volume: 2

Pages: 529-531

Congress: Jornadas sobre producción animal (15. 2013. Zaragoza)

Type: Conference paper

Abstract

This study aimed at identifying genetic markers associated with beef tenderness in two largely different muscles, Psoas major (PM) en Flexor digitorum (FD). A total of 397 Avileña-Negra Ibérica calves with genotypes for the Illumina Bovine SNP50 platform and measures of organoleptic tenderness (OT) and Warner-Blazter compression (WBC) participated in the study. A pre-selection of SNPs was carried out by determining the SNPs contained in 140 QTL regions previously identified as associated with beef tenderness in the Animal Genome UMD_3.1 database. A genome wide association study (GWAS) was then performed using QXpak 5.2. A model including place-year and length of fattening, age, place and season of slaughter, panel session and panelist (only for OT), one SNP at a time and the polygenic additive effect was used. A total of 4101 SNPs were pre-selected. The GWAS revealed that 3/0 and 5/0 SNPs were associated to OT and WBC in FD/PM for a 12 and a 7 % FDR threshold, respectively. The SNP of largest effect was responsible for a of 7% and 32% of the total additive genetic variance of OT and WBC inFD, respectively. These results suggest a differential pattern of genetic regulation of tenderness between the two muscles