César Roncero Maíllo-rekin lankidetzan egindako argitalpenak (34)

2019

  1. c-Met Signaling Is Essential for Mouse Adult Liver Progenitor Cells Expansion After Transforming Growth Factor-β-Induced Epithelial–Mesenchymal Transition and Regulates Cell Phenotypic Switch

    Stem Cells, Vol. 37, Núm. 8, pp. 1108-1118

2018

  1. Bone morphogenetic protein 9 as a key regulator of liver progenitor cells in DDC-induced cholestatic liver injury

    Liver International, Vol. 38, Núm. 9, pp. 1664-1675

2016

  1. Dissecting the role of epidermal growth factor receptor catalytic activity during liver regeneration and hepatocarcinogenesis

    Hepatology, Vol. 63, Núm. 2, pp. 604-619

  2. Tetrahydroisoquinoline-Derived Urea and 2,5-Diketopiperazine Derivatives as Selective Antagonists of the Transient Receptor Potential Melastatin 8 (TRPM8) Channel Receptor and Antiprostate Cancer Agents

    Journal of Medicinal Chemistry, Vol. 59, Núm. 12, pp. 5661-5683

2015

  1. BMP9-induced survival effect in liver tumor cells requires p38MAPK activation

    International Journal of Molecular Sciences, Vol. 16, Núm. 9, pp. 20431-20448

  2. HGF/c-Met signaling promotes liver progenitor cell migration and invasion by an epithelial-mesenchymal transition-independent, phosphatidyl inositol-3 kinase-dependent pathway in an in vitro model

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1853, Núm. 10, pp. 2453-2463

  3. p38 MAPK down-regulates fibulin 3 expression through methylation of gene regulatory sequences: Role in migration and invasion

    Journal of Biological Chemistry, Vol. 290, Núm. 7, pp. 4383-4397

2013

  1. BMP9 Is a Proliferative and Survival Factor for Human Hepatocellular Carcinoma Cells

    PLoS ONE, Vol. 8, Núm. 7

  2. Mouse Hepatic Oval Cells Require Met-Dependent PI3K to Impair TGF-β-Induced Oxidative Stress and Apoptosis

    PLoS ONE, Vol. 8, Núm. 1

2012

  1. EGFR is dispensable for c-Met-mediated proliferation and survival activities in mouse adult liver oval cells

    Cellular Signalling, Vol. 24, Núm. 2, pp. 505-513

  2. NADPH Oxidase NOX4 Mediates Stellate Cell Activation and Hepatocyte Cell Death during Liver Fibrosis Development

    PLoS ONE, Vol. 7, Núm. 9

2008

  1. Upregulation of the NADPH oxidase NOX4 by TGF-beta in hepatocytes is required for its pro-apoptotic activity

    Journal of Hepatology, Vol. 49, Núm. 6, pp. 965-976

2007

  1. Activation of NADPH oxidase by transforming growth factor-β in hepatocytes mediates up-regulation of epidermal growth factor receptor ligands through a nuclear factor-κB-dependent mechanism

    Biochemical Journal, Vol. 405, Núm. 2, pp. 251-259

  2. Survival and apoptosis: A dysregulated balance in liver cancer

    Liver International, Vol. 27, Núm. 2, pp. 155-162

2006

  1. EGF blocks NADPH oxidase activation by TGF-β in fetal rat hepatocytes, impairing oxidative stress, and cell death

    Journal of Cellular Physiology, Vol. 207, Núm. 2, pp. 322-330

2002

  1. Differential role of PPARγ in the regulation of UCP-1 and adipogenesis by TNF-α in brown adipocytes

    FEBS Letters, Vol. 520, Núm. 1-3, pp. 58-62

2001

  1. Activation of caspases occurs downstream from radical oxygen species production, Bcl-xL down-regulation, and early cytochrome c release in apoptosis induced by transforming growth factor β in rat fetal hepatocytes

    Hepatology, Vol. 34, Núm. 3, pp. 548-556

  2. Activation of p38MAPK by TGF-β in fetal rat hepatocytes requires radical oxygen production, but is dispensable for cell death

    FEBS Letters, Vol. 499, Núm. 3, pp. 225-229

  3. Reactive oxygen species (ROS) mediates the mitochondrial-dependent apoptosis induced by transforming growth factor β in fetal hepatocytes

    FASEB Journal, Vol. 15, Núm. 3, pp. 741-751

  4. TNF-α inhibits UCP-1 expression in brown adipocytes via ERKs - Opposite effect of p38MAPK

    FEBS Letters, Vol. 493, Núm. 1, pp. 6-11