Publicaciones en colaboración con investigadores/as de Institut d'Investigacions Biomèdiques August Pi i Sunyer (16)

2022

  1. Aberrant TIMP-1 overexpression in tumor-associated fibroblasts drives tumor progression through CD63 in lung adenocarcinoma

    Matrix Biology, Vol. 111, pp. 207-225

  2. Mitochondrial bioenergetics boost macrophage activation, promoting liver regeneration in metabolically compromised animals

    Hepatology, Vol. 75, Núm. 3, pp. 550-566

  3. Semaphorin-3F/Neuropilin-2 Transcriptional Expression as a Predictive Biomarker of Occult Lymph Node Metastases in HNSCC

    Cancers, Vol. 14, Núm. 9

2021

  1. Anti-TGFβ (Transforming Growth Factor β) Therapy With Betaglycan-Derived P144 Peptide Gene Delivery Prevents the Formation of Aortic Aneurysm in a Mouse Model of Marfan Syndrome

    Arteriosclerosis, Thrombosis, and Vascular Biology, Vol. 41, Núm. 9, pp. E440-E452

  2. C3G downregulation induces the acquisition of a mesenchymal phenotype that enhances aggressiveness of glioblastoma cells

    Cell Death and Disease, Vol. 12, Núm. 4

2020

  1. Breast mammographic density: Stromal implications on breast cancer detection and therapy

    Journal of Clinical Medicine, Vol. 9, Núm. 3

  2. Epigenetic SmAd3 repression in tumor-associated fibroblasts impairs fibrosis and response to the antifibrotic drug nintedanib in lung squamous cell carcinoma

    Cancer Research, Vol. 80, Núm. 2, pp. 276-290

  3. The novel proautophagy anticancer drug ABTL0812 potentiates chemotherapy in adenocarcinoma and squamous nonsmall cell lung cancer

    International Journal of Cancer, Vol. 147, Núm. 4, pp. 1163-1179

  4. Tumor-associated fibroblasts promote HER2-targeted therapy resistance through FGFR2 activation

    Clinical Cancer Research, Vol. 26, Núm. 6, pp. 1432-1448

2018

  1. Altered TGF-β endocytic trafficking contributes to the increased signaling in Marfan syndrome

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1864, Núm. 2, pp. 554-562

  2. Glucocorticoids promote transition of ductal carcinoma in situ to invasive ductal carcinoma by inducing myoepithelial cell apoptosis

    Breast Cancer Research, Vol. 20, Núm. 1

  3. Histamine receptor 1 inhibition enhances antitumor therapeutic responses through extracellular signal-regulated kinase (ERK) activation in breast cancer

    Cancer Letters, Vol. 424, pp. 70-83

2016

  1. C3G knock-down enhances migration and invasion by increasing Rap1-mediated p38α activation, while it impairs tumor growth through p38α-independent mechanisms

    Oncotarget, Vol. 7, Núm. 29, pp. 45060-45078

  2. Differential expression of neurogenes among breast cancer subtypes identifies high risk patients

    Oncotarget, Vol. 7, Núm. 5, pp. 5313-5326

2015

  1. Vascular smooth muscle cell phenotypic changes in patients with marfan syndrome

    Arteriosclerosis, Thrombosis, and Vascular Biology, Vol. 35, Núm. 4, pp. 960-972

2014

  1. Mechanisms of disseminated cancer cell dormancy: An awakening field

    Nature Reviews Cancer, Vol. 14, Núm. 9, pp. 611-622