Publikationen in Zusammenarbeit mit Forschern von Institut d'Investigació Biomédica de Bellvitge (77)

2024

  1. Iron chelation as a new therapeutic approach to prevent senescence and liver fibrosis progression

    Cell Death and Disease, Vol. 15, Núm. 9

  2. New Insights into the Exosome-Induced Migration of Uveal Melanoma Cells and the Pre-Metastatic Niche Formation in the Liver

    Cancers, Vol. 16, Núm. 17

  3. The hepatocyte epidermal growth factor receptor (EGFR) pathway regulates the cellular interactome within the liver fibrotic niche

    Journal of Pathology, Vol. 263, Núm. 4-5, pp. 482-495

  4. Uncovering a Novel Functional Interaction Between Adult Hepatic Progenitor Cells, Inflammation and EGFR Signaling During Bile Acids-Induced Injury

    International Journal of Biological Sciences, Vol. 20, Núm. 7, pp. 2339-2355

2023

  1. Autotaxin inhibitor IOA-289 reduces gastrointestinal cancer progression in preclinical models

    Journal of Experimental and Clinical Cancer Research, Vol. 42, Núm. 1

  2. Correction: Autotaxin inhibitor IOA-289 reduces gastrointestinal cancer progression in preclinical models (Journal of Experimental & Clinical Cancer Research, (2023), 42, 1, (197), 10.1186/s13046-023-02780-4)

    Journal of Experimental and Clinical Cancer Research

  3. Dissecting the role of the NADPH oxidase NOX4 in TGF-beta signaling in hepatocellular carcinoma

    Redox Biology, Vol. 65

  4. The NADPH oxidase NOX4 regulates redox and metabolic homeostasis preventing HCC progression

    Hepatology, Vol. 78, Núm. 2, pp. 416-433

2022

  1. Lack of EGFR catalytic activity in hepatocytes improves liver regeneration following DDC-induced cholestatic injury by promoting a pro-restorative inflammatory response

    Journal of Pathology, Vol. 258, Núm. 3, pp. 312-324

  2. Mitochondrial bioenergetics boost macrophage activation, promoting liver regeneration in metabolically compromised animals

    Hepatology, Vol. 75, Núm. 3, pp. 550-566

2021

  1. 2-[18f]fdg pet/ct as a predictor of microvascular invasion and a high histological grade in patients with a hepatocellular carcinoma

    Cancers, Vol. 13, Núm. 11

  2. Anti-miR-518d-5p overcomes liver tumor cell death resistance through mitochondrial activity

    Cell Death and Disease, Vol. 12, Núm. 6

  3. Epithelial–mesenchymal transition (Emt) induced by tgf-β in hepatocellular carcinoma cells reprograms lipid metabolism

    International Journal of Molecular Sciences, Vol. 22, Núm. 11

  4. The tgf-β /nadph oxidases axis in the regulation of liver cell biology in health and disease

    Cells, Vol. 10, Núm. 9

  5. The tgf-β pathway: A pharmacological target in hepatocellular carcinoma?

    Cancers, Vol. 13, Núm. 13

2020

  1. Clathrin switches transforming growth factor-β role to pro-tumorigenic in liver cancer

    Journal of Hepatology, Vol. 72, Núm. 1, pp. 125-134

2019

  1. Calcium regulates HCC proliferation as well as EGFR recycling/degradation and could be a new therapeutic target in HCC

    Cancers, Vol. 11, Núm. 10

  2. Paradoxical role of the NADPH oxidase NOX4 in early preneoplastic stages of hepatocytes induced by amino acid deprivation

    Biochimica et Biophysica Acta - General Subjects, Vol. 1863, Núm. 4, pp. 714-722

  3. c-Met Signaling Is Essential for Mouse Adult Liver Progenitor Cells Expansion After Transforming Growth Factor-β-Induced Epithelial–Mesenchymal Transition and Regulates Cell Phenotypic Switch

    Stem Cells, Vol. 37, Núm. 8, pp. 1108-1118

2018

  1. Altered TGF-β endocytic trafficking contributes to the increased signaling in Marfan syndrome

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1864, Núm. 2, pp. 554-562