Mecanismos de señalización celular en enfermedades hepáticas, cardiovasculares y cáncer
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Centro de Investigacion Biomedica en Red de Diabetes y Enfermedades Metabolicas Asociadas
Madrid, EspañaPublicaciones en colaboración con investigadores/as de Centro de Investigacion Biomedica en Red de Diabetes y Enfermedades Metabolicas Asociadas (10)
2024
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The hepatocyte epidermal growth factor receptor (EGFR) pathway regulates the cellular interactome within the liver fibrotic niche
Journal of Pathology, Vol. 263, Núm. 4-5, pp. 482-495
2023
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The NADPH oxidase NOX4 regulates redox and metabolic homeostasis preventing HCC progression
Hepatology, Vol. 78, Núm. 2, pp. 416-433
2021
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Epithelial–mesenchymal transition (Emt) induced by tgf-β in hepatocellular carcinoma cells reprograms lipid metabolism
International Journal of Molecular Sciences, Vol. 22, Núm. 11
2020
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C3G contributes to platelet activation and aggregation by regulating major signaling pathways
Signal Transduction and Targeted Therapy, Vol. 5, Núm. 1
2018
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Dual role of protein tyrosine phosphatase 1B in the progression and reversion of non-alcoholic steatohepatitis
Molecular Metabolism, Vol. 7, pp. 132-146
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TWEAK promotes migration and invasion in MEFs through a mechanism dependent on ERKs activation and Fibulin 3 down-regulation
Journal of Cellular Physiology, Vol. 233, Núm. 2, pp. 968-978
2017
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Role of the Transforming Growth Factor-β in regulating hepatocellular carcinoma oxidative metabolism
Scientific Reports, Vol. 7, Núm. 1
2016
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Dissecting the role of epidermal growth factor receptor catalytic activity during liver regeneration and hepatocarcinogenesis
Hepatology, Vol. 63, Núm. 2, pp. 604-619
2013
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TWEAK prevents TNF-α-induced insulin resistance through PP2A activation in human adipocytes
American Journal of Physiology - Endocrinology and Metabolism, Vol. 305, Núm. 1
2012
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Protein-tyrosine phosphatase 1B (PTP1B) deficiency confers resistance to transforming growth factor-β (TGF-β)-induced suppressor effects in hepatocytes
Journal of Biological Chemistry, Vol. 287, Núm. 19, pp. 15263-15274