David Bartolomé Martín-rekin lankidetzan egindako argitalpenak (14)

2020

  1. β-adrenergic receptors/epac signaling increases the size of the readily releasable pool of synaptic vesicles required for parallel fiber LTP

    Journal of Neuroscience, Vol. 40, Núm. 45, pp. 8604-8617

2019

  1. Photoconversion of FM1-43 Reveals Differences in Synaptic Vesicle Recycling and Sensitivity to Pharmacological Disruption of Actin Dynamics in Individual Synapses

    ACS Chemical Neuroscience, Vol. 10, Núm. 4, pp. 2045-2059

2017

  1. CB1 receptors down-regulate a cAMP/Epac2/PLC pathway to silence the nerve terminals of cerebellar granule cells

    Journal of Neurochemistry, Vol. 142, Núm. 3, pp. 350-364

2016

  1. Cross-talk between metabotropic glutamate receptor 7 and beta adrenergic receptor signaling at cerebrocortical nerve terminals

    Neuropharmacology, Vol. 101, pp. 412-425

2014

  1. Cannabinoid type 1 receptors transiently silence glutamatergic nerve terminals of cultured cerebellar granule cells

    PLoS ONE, Vol. 9, Núm. 2

2013

  1. Potentiation of mGlu7 receptor-mediated glutamate release at nerve terminals containing N and P/Q type Ca2+ channels

    Neuropharmacology, Vol. 67, pp. 213-222

  2. Studying synaptic efficiency by post-hoc immunolabelling

    BMC Neuroscience, Vol. 14

  3. β-adrenergic receptors activate exchange protein directly activated by cAMP (Epac), translocate munc13-1, and enhance the Rab3A-RIM1 α interaction to potentiate glutamate release at cerebrocortical nerve terminals

    Journal of Biological Chemistry, Vol. 288, Núm. 43, pp. 31370-31385

2012

  1. Daidzein has neuroprotective effects through ligand-binding-independent PPARγ activation

    Neurochemistry International, Vol. 61, Núm. 1, pp. 119-127

  2. Efficient synaptic vesicle recycling after intense exocytosis concomitant with the accumulation of non-releasable endosomes at early developmental stages

    Journal of Cell Science, Vol. 125, Núm. 2, pp. 422-434

2011

  1. Non-additive potentiation of glutamate release by phorbol esters and metabotropic mGlu7 receptor in cerebrocortical nerve terminals

    Journal of Neurochemistry, Vol. 116, Núm. 4, pp. 476-485

2009

  1. Partial compensation for N-type Ca2+ channel loss by P/Q-type Ca2+ channels underlines the differential release properties supported by these channels at cerebrocortical nerve terminals

    European Journal of Neuroscience, Vol. 29, Núm. 6, pp. 1131-1140

  2. Suppression of guanylyl cyclase (β1 subunit) expression impairs neurite outgrowth and synapse maturation in cultured cerebellar granule cells

    Cell Death and Differentiation, Vol. 16, Núm. 9, pp. 1266-1278

2008

  1. The inhibition of release by mGlu7 receptors is independent of the Ca2+ channel type but associated to GABAB and adenosine A1 receptors

    Neuropharmacology, Vol. 55, Núm. 4, pp. 464-473