Publikationen, an denen er mitarbeitet INÉS SÁNCHEZ-ROMÁN ROJAS (15)

2023

  1. USE OF ONLINE EDUCATIVE DIGITAL TOOLS TO ENCOURAGE PARTICIPATION AND ACTIVE LEARNING OF ANIMAL PHYSIOLOGY (DIGIFISIO)

    ICERI2023 Proceedings

2020

  1. Mitochondrial base excision repair positively correlates with longevity in the liver and heart of mammals

    GeroScience, Vol. 42, Núm. 2, pp. 653-665

2014

  1. Independent and additive effects of atenolol and methionine restriction on lowering rat heart mitochondria oxidative stress

    Journal of Bioenergetics and Biomembranes, Vol. 46, Núm. 3, pp. 159-172

  2. Lifelong treatment with atenolol decreases membrane fatty acid unsaturation and oxidative stress in heart and skeletal muscle mitochondria and improves immunity and behavior, without changing mice longevity

    Aging Cell, Vol. 13, Núm. 3, pp. 551-560

2013

  1. Reduction in mitochondrialmembrane peroxidizability index and protein lipoxidation levels in therat heart after ß-adrenergic receptor signaling interruption with the ß-blocker atenolol

    Anales de la Real Academia Nacional de Farmacia, Núm. 4, pp. 613-633

2012

  1. Effects of aging and methionine restriction applied at old age on ROS generation and oxidative damage in rat liver mitochondria

    Biogerontology, Vol. 13, Núm. 4, pp. 399-411

2011

  1. Forty percent methionine restriction lowers DNAmethylation, complex i ROS generation, and oxidative damage to mtDNA and mitochondrial proteins in rat heart

    Journal of Bioenergetics and Biomembranes, Vol. 43, Núm. 6, pp. 699-708

  2. Methionine and homocysteine modulate the rate of ROS generation of isolated mitochondria in vitro

    Journal of Bioenergetics and Biomembranes, Vol. 43, Núm. 4, pp. 377-386

2010

  1. The β-blocker atenolol lowers the longevity-related degree of fatty acid unsaturation, decreases protein oxidative damage, and increases extracellular signal-regulated kinase signaling in the heart of C57BL/6 mice

    Rejuvenation Research, Vol. 13, Núm. 6, pp. 683-693

2009

  1. Efecto de la restricción de aminoácidos excepto la metionina sobre el estrés oxidativo mitocondrial

    Revista Espanola de Geriatria y Gerontologia, Vol. 44, Núm. 4, pp. 194-199

  2. Effect of 40% restriction of dietary amino acids (except methionine) on mitochondrial oxidative stress and biogenesis, AIF and SIRT1 in rat liver

    Biogerontology, Vol. 10, Núm. 5, pp. 579-592

  3. Effect of methionine dietary supplementation on mitochondrial oxygen radical generation and oxidative DNA damage in rat liver and heart

    Journal of Bioenergetics and Biomembranes, Vol. 41, Núm. 3, pp. 309-321

  4. Forty percent methionine restriction decreases mitochondrial oxygen radical production and leak at complex I during forward electron flow and lowers oxidative damage to proteins and mitochondrial DNA in rat kidney and brain mitochondria.

    Rejuvenation research, Vol. 12, Núm. 6, pp. 421-434

2008

  1. Effect of every other day feeding on mitochondrial free radical production and oxidative stress in mouse liver

    Rejuvenation Research, Vol. 11, Núm. 3, pp. 621-629

  2. Forty percent and eighty percent methionine restriction decrease mitochondrial ROS generation and oxidative stress in rat liver

    Biogerontology, Vol. 9, Núm. 3, pp. 183-196