Publicaciones en colaboración con investigadores/as de Hospital Universitario de la Princesa (45)

2024

  1. A New Method to Evaluate Lower Esophageal Distension Capacity in Eosinophilic Esophagitis by Using Functional Lumen Imaging Probe (EndoFLIP™)

    Diagnostics, Vol. 14, Núm. 2

2023

  1. Proteomic analysis of the esophageal epithelium reveals key features of eosinophilic esophagitis pathophysiology

    Allergy: European Journal of Allergy and Clinical Immunology, Vol. 78, Núm. 10, pp. 2732-2744

2021

  1. Poor sensitivity of fecal gluten immunogenic peptides and serum antibodies to detect duodenal mucosal damage in celiac disease monitoring

    Nutrients, Vol. 13, Núm. 1, pp. 1-12

  2. Proton pump inhibitor therapy reverses endoscopic features of fibrosis in eosinophilic esophagitis

    Digestive and Liver Disease, Vol. 53, Núm. 11, pp. 1479-1485

2020

  1. Naturally occurring and engineered alphaviruses sensitive to double-stranded-RNA-activated protein kinase show restricted translation in mammalian cells, increased sensitivity to interferon, and marked oncotropism

    Journal of Virology, Vol. 94, Núm. 3

2017

  1. Corrigendum to "T Helper 17/Regulatory T Cell Balance and Experimental Models of Peritoneal Dialysis-Induced Damage"

    BioMed research international

  2. Polyanionic carbosilane dendrimers prevent hepatitis C virus infection in cell culture

    Nanomedicine: Nanotechnology, Biology, and Medicine, Vol. 13, Núm. 1, pp. 49-58

2016

  1. Mesothelial-to-mesenchymal transition in the pathogenesis of post-surgical peritoneal adhesions

    Journal of Pathology, Vol. 239, Núm. 1, pp. 48-59

2015

  1. Apolipoprotein E, but not apolipoprotein B, is essential for efficient cell-to-cell transmission of hepatitis C virus

    Journal of Virology, Vol. 89, Núm. 19, pp. 9962-9973

  2. Clathrin mediates infectious hepatitis C virus particle egress

    Journal of Virology, Vol. 89, Núm. 8, pp. 4180-4190

  3. T helper 17/regulatory t cell balance and experimental models of peritoneal dialysis-induced damage

    BioMed Research International, Vol. 2015

2014

  1. Apicobasal polarity controls lymphocyte adhesion to hepatic epithelial cells

    Cell Reports, Vol. 8, Núm. 6, pp. 1879-1893

  2. Modulation of PI3K-LXRα-dependent lipogenesis mediated by oxidative/nitrosative stress contributes to inhibition of HCV replication by quercetin

    Laboratory Investigation, Vol. 94, Núm. 3, pp. 262-274

2013

  1. Carcinoma-associated fibroblasts derive from mesothelial cells via mesothelial-to-mesenchymal transition in peritoneal metastasis

    Journal of Pathology, Vol. 231, Núm. 4, pp. 517-531

  2. Functional Relevance of the Switch of VEGF Receptors/Co-Receptors during Peritoneal Dialysis-Induced Mesothelial to Mesenchymal Transition

    PLoS ONE, Vol. 8, Núm. 4

  3. Resumen de congreso: 20 años después del primer Simposium Internacional sobre el virus de la hepatitis C y otros virus relacionados

    Gastroenterologia y Hepatologia, Vol. 36, Núm. 10, pp. 641-646

2012

  1. Liver X receptor α-mediated regulation of lipogenesis by core and NS5A proteins contributes to HCV-induced liver steatosis and HCV replication

    Laboratory Investigation, Vol. 92, Núm. 8, pp. 1191-1202

  2. Matrigel-embedded 3D culture of Huh-7 cells as a hepatocyte-like polarized system to study hepatitis C virus cycle

    Virology, Vol. 425, Núm. 1, pp. 31-39

  3. Role of tight junctions in hepatitis C virus infection.

    Revista española de enfermedades digestivas : organo oficial de la Sociedad Española de Patología Digestiva, Vol. 104, Núm. 5, pp. 255-263

2011

  1. Blocking TGF-β1 protects the peritoneal membrane from dialysate-induced damage

    Journal of the American Society of Nephrology, Vol. 22, Núm. 9, pp. 1682-1695