Fundación Jiménez Díaz -ko ikertzaileekin lankidetzan egindako argitalpenak (24)

1997

  1. The 80-kD fibronectin fragment increases the production of fibronectin and tumour necrosis factor-alpha (TNF-α) in cultured mesangial cells

    Clinical and Experimental Immunology, Vol. 107, Núm. 2, pp. 398-403

1996

  1. Anti-inflammatory effect of a PAF receptor antagonist and a new molecule with antiproteinase activity in an experimental model of acute urate crystal arthritis

    Journal of Lipid Mediators and Cell Signalling, Vol. 13, Núm. 1, pp. 35-49

  2. Interferon-inducible protein-10 is highly expressed in rats with experimental nephrosis

    American Journal of Pathology, Vol. 148, Núm. 1, pp. 301-311

  3. Urinary excretion of platelet-activating factor in human and experimental nephrosis

    Nephrology Dialysis Transplantation, Vol. 11, Núm. 2, pp. 282-286

1995

  1. Fibronectin (FN) decreases glomerular lesions and synthesis of tumour necrosis factor-alpha (TNF-α), platelet-activating factor (PAF) and FN in proliferative glomerulonephritis

    Clinical and Experimental Immunology, Vol. 101, Núm. 2, pp. 334-340

  2. IL-1β and IL-6 stimulate the production of platelet-activating factor (PAF) by cultured rabbit synovial cells

    Clinical and Experimental Immunology, Vol. 99, Núm. 3, pp. 364-368

1994

  1. Increase of tumour necrosis factor α synthesis and gene expression in peripheral blood mononuclear cells of children with idiopathic nephrotic syndrome

    European Journal of Clinical Investigation, Vol. 24, Núm. 12, pp. 799-805

  2. Involvement of tumor necrosis factor and platelet-activating factor in the pathogenesis of experimental nephrosis in rats

    Laboratory Investigation, Vol. 70, Núm. 4, pp. 449-459

  3. Long-term effect of nonsteroidal anti-inflammatory drugs on the production of cytokines and other inflammatory mediators by blood cells of patients with osteoarthritis

    Agents and Actions, Vol. 41, Núm. 3-4, pp. 171-178

  4. Soluble IgA and IgG aggregates are catabolized by cultured rat mesangial cells and induce production of TNF-α and IL-6, and proliferation

    Journal of Immunology, Vol. 153, Núm. 11, pp. 5247-5256

1993

  1. Evidence for a specific IgA receptor in rat and human mesangial cells

    Journal of Immunology, Vol. 151, Núm. 12, pp. 7172-7181

  2. Role of tumor necrosis factor-α in the pathogenesis of glomerular diseases

    Kidney International, Supplement

  3. The intercrine superfamily and renal disease

    Kidney International, Supplement

1992

  1. IL‐1‐like production in adriamycin‐induced nephrotic syndrome in the rat

    Clinical & Experimental Immunology, Vol. 87, Núm. 1, pp. 117-121

  2. Involvement of platelet‐activating factor and tumour necrosis factor in the pathogenesis of joint inflammation in rabbits

    Clinical & Experimental Immunology, Vol. 88, Núm. 2, pp. 318-323

  3. LA PARTICIPACION DE CITOQUINAS Y MATRIZ EXTRACELULAR EN LA PROGRESION DEL DANO RENAL

    Nefrologia, Vol. 12, Núm. SUPPL. 5, pp. 22-32

1991

  1. Involvement of lipid mediators in the pathogenesis of experimental nephrosis in rats: Its pharmacological modulation

    Renal Failure, Vol. 13, Núm. 2-3, pp. 95-101

  2. The role of platelet-activating factor (PAF) in experimental glomerular injury

    Lipids, Vol. 26, Núm. 12, pp. 1310-1315

1990

  1. Detection and characterization of circulating and glomerular immune complexes in experimental IgA nephropathy

    Immunology, Vol. 70, Núm. 3, pp. 296-302

  2. Rational bases for the use of cyclosporin A in the nephrotic syndrome

    Nefrologia, Vol. 10, Núm. SUPPL. 5, pp. 16-24