Publicaciones en las que colabora con MARGARITA LORENZO BALADO (31)

2005

  1. Cellular Models for the Study of Type 2 Diabetes

    The Metabolic Syndrome at the Beginning of the XXI Century: A Genetic and Molecular Approach (Elsevier Inc.), pp. 43-65

  2. The brown adipose cell: A model for understanding the molecular mechanisms of insulin resistance

    Acta Physiologica Scandinavica, Vol. 183, Núm. 1, pp. 59-73

2001

  1. Insulin produces myogenesis in C2C12 myoblasts by induction of NF-κB and downregulation of AP-1 activities

    Journal of Cellular Physiology, Vol. 186, Núm. 1, pp. 82-94

2000

  1. Akt mediates insulin rescue from apoptosis in brown adipocytes: Effect of ceramide

    Growth Hormone and IGF Research, Vol. 10, Núm. 5, pp. 256-266

  2. Okadaic acid inhibits insulin-induced glucose transport in fetal brown adipocytes in an Akt-independent and protein kinase C ζ-dependent manner

    FEBS Letters, Vol. 472, Núm. 1, pp. 153-158

1999

  1. Activated Ha-ras induces apoptosis by association with phosphorylated Bcl-2 in a mitogen-activated protein kinase-independent manner

    Journal of Biological Chemistry, Vol. 274, Núm. 27, pp. 18857-18863

  2. Inhibition of caspases rescues brown adipocytes from apoptosis downregulating BCL-XS and upregulating BCL-2 gene expression

    Experimental Cell Research, Vol. 246, Núm. 2, pp. 301-307

  3. Insulin and insulin-like growth factor I up-regulate GLUT4 gene expression in fetal brown adipocytes, in a phosphoinositide 3-kinase-dependent manner

    Biochemical Journal, Vol. 337, Núm. 3, pp. 397-405

1998

  1. H-ras induces glucose uptake in brown adipocytes in an insulin- and phosphatidylinositol 3-kinase-independent manner

    Experimental Cell Research, Vol. 243, Núm. 2, pp. 274-281

  2. Inhibition of PI 3-kinase and RAS blocks IGF-I and insulin-induced uncoupling protein 1 gene expression in brown adipocytes

    Journal of Cellular Physiology, Vol. 176, Núm. 1, pp. 99-109

  3. Insulin receptor substrate (IRS) proteins IRS-1 and IRS-2 differential signaling in the insulin/insulin-like growth factor-I pathways in fetal brown adipocytes

    Molecular Endocrinology, Vol. 12, Núm. 5, pp. 688-697

  4. Insulin/IGF-I rescues immortalized brown adipocytes from apoptosis down- regulating Bcl-xS expression, in a PI 3-kinase- and map kinase-dependent manner

    Experimental Cell Research, Vol. 243, Núm. 2, pp. 213-221

  5. Tumor necrosis factor-α causes insulin receptor substrate-2-mediated insulin resistance and inhibits insulin-induced adipogenesis in fetal brown adipocytes

    Endocrinology, Vol. 139, Núm. 3, pp. 1229-1238

1997

  1. Alterations in the insulin signaling pathway induced by immortalization and H-ras transformation of brown adipocytes

    Endocrinology, Vol. 138, Núm. 8, pp. 3195-3206

  2. Malic enzyme and glucose 6-phosphate dehydrogenase gene expression increases in rat liver cirrhogenesis

    British Journal of Cancer, Vol. 75, Núm. 4, pp. 487-492

  3. Phosphatidylinositol 3-kinase is a requirement for insulin-like growth factor I-induced differentiation, but not for mitogenesis, in fetal brown adipocytes

    Molecular Endocrinology, Vol. 11, Núm. 5, pp. 595-607

1996

  1. IGF-I: A Mitogen also involved in differentiation processes in mammalian cells

    International Journal of Biochemistry and Cell Biology, Vol. 28, Núm. 5, pp. 499-510

  2. Insulin-like growth factor I and insulin induce adipogenic-related gene expression in fetal brown adipocyte primary cultures

    Biochemical Journal, Vol. 319, Núm. 2, pp. 627-632

  3. Involvement of Raf-1 kinase and protein kinase C ζ in insulin-like growth factor I-induced brown adipocyte mitogenic signaling cascades: Inhibition by cyclic adenosine 3',5'-monophosphate

    Endocrinology, Vol. 137, Núm. 9, pp. 3832-3841

  4. P-3: IGF-I/RAS induces growth and differentiation in brown adipocytes: A novel thermogenic mechanism

    Experimental and Clinical Endocrinology and Diabetes, Vol. 104, pp. 67-68