Publications by the researcher in collaboration with María Carmen Guerrero Arroyo (22)

2025

  1. C3G promotes bone marrow adipocyte expansion and hematopoietic regeneration after myeloablation by enhancing megakaryocyte niche function

    Journal of Hematology and Oncology, Vol. 18, Núm. 1

  2. Correction: Deletion of C3G in hepatocytes impairs full liver maturation and alters glucose homeostasis (Cell Death & Disease, (2025), 16, 1, (711), 10.1038/s41419-025-08031-y)

    Cell Death and Disease

  3. Deletion of C3G in hepatocytes impairs full liver maturation and alters glucose homeostasis

    Cell Death and Disease, Vol. 16, Núm. 1

  4. Platelet C3G protects from liver fibrosis, while enhancing tumor growth through regulation of the immune response

    Journal of Pathology, Vol. 265, Núm. 4, pp. 502-517

2024

  1. Platelet C3G: a key player in vesicle exocytosis, spreading and clot retraction

    Cellular and Molecular Life Sciences, Vol. 81, Núm. 1

2022

  1. C3G down-regulation enhances pro-migratory and stemness properties of oval cells by promoting an epithelial-mesenchymal-like process

    International journal of biological sciences, Vol. 18, Núm. 15, pp. 5873-5884

  2. New functions of C3G in platelet biology: Contribution to ischemia-induced angiogenesis, tumor metastasis and TPO clearance

    Frontiers in Cell and Developmental Biology, Vol. 10

2021

  1. C3G downregulation induces the acquisition of a mesenchymal phenotype that enhances aggressiveness of glioblastoma cells

    Cell Death and Disease, Vol. 12, Núm. 4

  2. C3G protein, a new player in glioblastoma

    International Journal of Molecular Sciences, Vol. 22, Núm. 18

  3. Contribution of c3g and other gefs to liver cancer development and progression

    Hepatoma Research, Vol. 7

2020

  1. C3G contributes to platelet activation and aggregation by regulating major signaling pathways

    Signal Transduction and Targeted Therapy, Vol. 5, Núm. 1

  2. C3G is upregulated in hepatocarcinoma, contributing to tumor growth and progression and to HGF/MET pathway activation

    Cancers, Vol. 12, Núm. 8, pp. 1-22

2018

  1. C3G, through its GEF activity, induces megakaryocytic differentiation and proplatelet formation

    Cell Communication and Signaling, Vol. 16, Núm. 1

  2. How Rap and its GEFs control liver physiology and cancer development. C3G alterations in human hepatocarcinoma

    HEPATIC ONCOLOGY, Vol. 5, Núm. 1

2017

  1. C3G promotes a selective release of angiogenic factors from activated mouse platelets to regulate angiogenesis and tumor metastasis

    Oncotarget, Vol. 8, Núm. 67, pp. 110994-111011

2016

  1. C3G knock-down enhances migration and invasion by increasing Rap1-mediated p38α activation, while it impairs tumor growth through p38α-independent mechanisms

    Oncotarget, Vol. 7, Núm. 29, pp. 45060-45078

2015

  1. p38 MAPK down-regulates fibulin 3 expression through methylation of gene regulatory sequences: Role in migration and invasion

    Journal of Biological Chemistry, Vol. 290, Núm. 7, pp. 4383-4397

2013

  1. C3G forms complexes with Bcr-Abl and p38α MAPK at the focal adhesions in chronic myeloid leukemia cells: Implication in the regulation of leukemic cell adhesion

    Cell Communication and Signaling, Vol. 11, Núm. 1

  2. Met signaling in cardiomyocytes is required for normal cardiac function in adult mice

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1832, Núm. 12, pp. 2204-2215

2012

  1. C3G transgenic mouse models with specific expression in platelets reveal a new role for C3G in platelet clotting through its GEF activity

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1823, Núm. 8, pp. 1366-1377