Publicacions en què col·labora amb MARGARITA LORENZO BALADO (55)

2009

  1. From insulin action to hormonal resistance: old to recent molecular mechanisms

    Type 2 diabetes mellitus (Elsevier), pp. 105-129

2005

  1. Cellular Models for the Study of Type 2 Diabetes

    The Metabolic Syndrome at the Beginning of the XXI Century: A Genetic and Molecular Approach (Elsevier Inc.), pp. 43-65

  2. The brown adipose cell: A model for understanding the molecular mechanisms of insulin resistance

    Acta Physiologica Scandinavica, Vol. 183, Núm. 1, pp. 59-73

2003

  1. Rosiglitazone and retinoic acid induce uncoupling protein-1 (UCP-1) in a p38 mitogen-activated protein kinase-dependent manner in fetal primary brown adipocytes

    Journal of Biological Chemistry, Vol. 278, Núm. 1, pp. 263-269

2002

  1. Insulin restores differentiation of Ras-transtormed C2C12 myoblasts by inducing NF-κB through an AKT/P70S6K/p38-MAPK pathway

    Oncogene, Vol. 21, Núm. 23, pp. 3739-3753

  2. PV-400 time-interval analyzing electro-optical transducer with wide-format photocathode and picosecond resolution time

    Measurement Techniques, Vol. 45, Núm. 4, pp. 374-376

2001

  1. Insulin produces myogenesis in C2C12 myoblasts by induction of NF-κB and downregulation of AP-1 activities

    Journal of Cellular Physiology, Vol. 186, Núm. 1, pp. 82-94

2000

  1. Akt mediates insulin rescue from apoptosis in brown adipocytes: Effect of ceramide

    Growth Hormone and IGF Research, Vol. 10, Núm. 5, pp. 256-266

  2. Apoptosis en adipocitos marrones: Implicación en obesidad y cáncer

    Anales de la Real Academia de Farmacia, Vol. 66, Núm. 3, pp. 313-334

  3. Apoptosis in Brown Adipocytes: Implication in Obesity and Cancer

    Anales de la Real Academia Nacional de Farmacia, Núm. 3, pp. 1

  4. Okadaic acid inhibits insulin-induced glucose transport in fetal brown adipocytes in an Akt-independent and protein kinase C ζ-dependent manner

    FEBS Letters, Vol. 472, Núm. 1, pp. 153-158

1999

  1. Activated Ha-ras induces apoptosis by association with phosphorylated Bcl-2 in a mitogen-activated protein kinase-independent manner

    Journal of Biological Chemistry, Vol. 274, Núm. 27, pp. 18857-18863

  2. Inhibition of caspases rescues brown adipocytes from apoptosis downregulating BCL-XS and upregulating BCL-2 gene expression

    Experimental Cell Research, Vol. 246, Núm. 2, pp. 301-307

  3. Insulin and insulin-like growth factor I up-regulate GLUT4 gene expression in fetal brown adipocytes, in a phosphoinositide 3-kinase-dependent manner

    Biochemical Journal, Vol. 337, Núm. 3, pp. 397-405

1998

  1. H-ras induces glucose uptake in brown adipocytes in an insulin- and phosphatidylinositol 3-kinase-independent manner

    Experimental Cell Research, Vol. 243, Núm. 2, pp. 274-281

  2. Inhibition of PI 3-kinase and RAS blocks IGF-I and insulin-induced uncoupling protein 1 gene expression in brown adipocytes

    Journal of Cellular Physiology, Vol. 176, Núm. 1, pp. 99-109

  3. Insulin receptor substrate (IRS) proteins IRS-1 and IRS-2 differential signaling in the insulin/insulin-like growth factor-I pathways in fetal brown adipocytes

    Molecular Endocrinology, Vol. 12, Núm. 5, pp. 688-697

  4. Insulin/IGF-I rescues immortalized brown adipocytes from apoptosis down- regulating Bcl-xS expression, in a PI 3-kinase- and map kinase-dependent manner

    Experimental Cell Research, Vol. 243, Núm. 2, pp. 213-221

  5. Tumor necrosis factor-α causes insulin receptor substrate-2-mediated insulin resistance and inhibits insulin-induced adipogenesis in fetal brown adipocytes

    Endocrinology, Vol. 139, Núm. 3, pp. 1229-1238

1997

  1. Alterations in the insulin signaling pathway induced by immortalization and H-ras transformation of brown adipocytes

    Endocrinology, Vol. 138, Núm. 8, pp. 3195-3206