Publicaciones en colaboración con investigadores/as de Centro de Investigacion Biomedica en Red de Diabetes y Enfermedades Metabolicas Asociadas (34)

2024

  1. The overexpression of human amylin in pancreatic β cells facilitate the appearance of amylin aggregates in the kidney contributing to diabetic nephropathy

    Scientific Reports, Vol. 14, Núm. 1

2021

  1. Biological actions and molecular mechanisms of sambucus Nigra L. In neurodegeneration: A cell culture approach

    Molecules, Vol. 26, Núm. 16

  2. Dietary polyphenols in metabolic and neurodegenerative diseases: Molecular targets in autophagy and biological effects

    Antioxidants, Vol. 10, Núm. 2, pp. 1-22

  3. Human amylin aggregates release within exosomes as a protective mechanism in pancreatic β cells: Pancreatic β-hippocampal cell communication

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1868, Núm. 5

  4. Insulin Resistance and Diabetes Mellitus in Alzheimer's Disease

    Cells, Vol. 10, Núm. 5

  5. Osteoporosis Remission and New Bone Formation with Mesoporous Silica Nanoparticles

    Advanced Science, Vol. 8, Núm. 16

  6. Severe Hepatic Insulin Resistance Induces Vascular Dysfunction: Improvement by Liver-Specific Insulin Receptor Isoform A Gene Therapy in a Murine Diabetic Model

    Cells, Vol. 10, Núm. 8

  7. Sirtuins in mechanistic target of rapamycin complex 1 signaling

    Sirtuin Biology in Medicine: Targeting New Avenues of Care in Development, Aging, and Disease (Elsevier), pp. 191-212

2020

  1. Specific knockout of p85α in brown adipose tissue induces resistance to high-fat diet–induced obesity and its metabolic complications in male mice

    Molecular Metabolism, Vol. 31, pp. 1-13

2019

  1. Essential role of glucokinase in the protection of pancreatic β cells to the glucose energetic status

    Cell Death Discovery, Vol. 5, Núm. 1

  2. Liver-specific insulin receptor isoform A expression enhances hepatic glucose uptake and ameliorates liver steatosis in a mouse model of diet-induced obesity

    DMM Disease Models and Mechanisms, Vol. 12, Núm. 2

2018

  1. MTORC1 overactivation as a key aging factor in the progression to type 2 diabetes mellitus

    Frontiers in Endocrinology, Vol. 9, Núm. OCT

  2. Pancreatic β cells overexpressing hIAPP impaired mitophagy and unbalanced mitochondrial dynamics

    Cell Death and Disease, Vol. 9, Núm. 5

  3. Potential role of insulin receptor isoforms and IGF receptors in plaque instability of human and experimental atherosclerosis

    Cardiovascular Diabetology, Vol. 17, Núm. 1

2017

  1. MTORC1 regulates both general autophagy and mitophagy induction after oxidative phosphorylation uncoupling

    Molecular and Cellular Biology, Vol. 37, Núm. 23

2016

  1. Expression of insulin receptor (IR) A and B isoforms, IGF-IR, and IR/IGF-IR hybrid receptors in vascular smooth muscle cells and their role in cell migration in atherosclerosis

    Cardiovascular Diabetology, Vol. 15, Núm. 1

  2. Insulin receptor isoform A ameliorates long-term glucose intolerance in diabetic mice

    DMM Disease Models and Mechanisms, Vol. 9, Núm. 11, pp. 1271-1281

  3. Prevalent role of the insulin receptor isoform A in the regulation of hepatic glycogen metabolism in hepatocytes and in mice

    Diabetologia, Vol. 59, Núm. 12, pp. 2702-2710

  4. TSC2 N-terminal lysine acetylation status affects to its stability modulating mTORC1 signaling and autophagy

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1863, Núm. 11, pp. 2658-2667

2015

  1. Insulin receptor isoform A confers a higher proliferative capability to pancreatic beta cells enabling glucose availability and IGF-I signaling

    Molecular and Cellular Endocrinology, Vol. 409, pp. 82-91