Publicacións nas que colabora con MERCEDES MUÑOZ PICOS (10)

2022

  1. Hyperoxaluria Induces Endothelial Dysfunction in Preglomerular Arteries: Involvement of Oxidative Stress

    Cells, Vol. 11, Núm. 15

2021

  1. Activation of AMP kinase ameliorates kidney vascular dysfunction, oxidative stress and inflammation in rodent models of obesity

    British Journal of Pharmacology, Vol. 178, Núm. 20, pp. 4085-4103

  2. Endothelial KCa1.1 and KCa3.1 channels mediate rat intrarenal artery endothelium-derived hyperpolarization response

    Acta Physiologica, Vol. 231, Núm. 4

2020

  1. Differential contribution of Nox1, Nox2 and Nox4 to kidney vascular oxidative stress and endothelial dysfunction in obesity

    Redox Biology, Vol. 28

  2. Nox1-derived oxidative stress as a common pathogenic link between obesity and hyperoxaluria-related kidney injury

    Urolithiasis, Vol. 48, Núm. 6, pp. 481-492

2018

  1. Impaired Ca2+ handling in resistance arteries from genetically obese Zucker rats: Role of the PI3K, ERK1/2 and PKC signaling pathways

    Biochemical Pharmacology, Vol. 152, pp. 114-128

2017

  1. CYP epoxygenase-derived H2O2 is involved in the endothelium-derived hyperpolarization (EDH) and relaxation of intrarenal arteries

    Free Radical Biology and Medicine, Vol. 106, pp. 168-183

2015

  1. COX-2 is involved in vascular oxidative stress and endothelial dysfunction of renal interlobar arteries from obese Zucker rats

    Free Radical Biology and Medicine, Vol. 84, pp. 77-90

2014

  1. Endothelin a (ETA) receptors are involved in augmented adrenergic vasoconstriction and blunted Nitric Oxide-Mediated Relaxation of Penile Arteries from Insulin-Resistant Obese Zucker Rats

    Journal of Sexual Medicine, Vol. 11, Núm. 6, pp. 1463-1474

  2. Endothelin-1 contributes to endothelial dysfunction and enhanced vasoconstriction through augmented superoxide production in penile arteries from insulin-resistant obese rats: Role of ETA and ETB receptors

    British Journal of Pharmacology, Vol. 171, Núm. 24, pp. 5682-5695