Publicaciones en las que colabora con PEDRO LORENZO FERNÁNDEZ (21)

2012

  1. Velázquez. Manual de farmacología básica y clínica

    Editorial Médica Panamericana

2007

  1. Ischemic preconditioning reveals that GLT1/EAAT2 glutamate transporter is a novel PPARγ target gene involved in neuroprotection

    Journal of Cerebral Blood Flow and Metabolism, Vol. 27, Núm. 7, pp. 1327-1338

  2. Toll-like receptor 4 is involved in brain damage and inflammation after experimental stroke

    Circulation, Vol. 115, Núm. 12, pp. 1599-1608

2006

  1. Neurorepair versus neuroprotection in stroke

    Cerebrovascular Diseases, Vol. 21, Núm. SUPPL. 2, pp. 54-63

  2. Rosiglitazone and 15-deoxy-Δ12,14-prostaglandin J 2 cause potent neuroprotection after experimental stroke through noncompletely overlapping mechanisms

    Journal of Cerebral Blood Flow and Metabolism, Vol. 26, Núm. 2, pp. 218-229

  3. TNFR1 mediates increased neuronal membrane EAAT3 expression after in vivo cerebral ischemic preconditioning

    Neuroscience, Vol. 138, Núm. 4, pp. 1171-1178

  4. Targets of cytoprotection in acute ischemic stroke: Present and future

    Cerebrovascular Diseases, Vol. 21, Núm. SUPPL. 2, pp. 1-8

2005

  1. Inhibition of iNOS activity by 1400W decreases glutamate release and ameliorates stroke outcome after experimental ischemia

    Neurobiology of Disease, Vol. 18, Núm. 2, pp. 375-384

  2. Neuroprotection afforded by prior citicoline administration in experimental brain ischemia: Effects on glutamate transport

    Neurobiology of Disease, Vol. 18, Núm. 2, pp. 336-345

  3. TNFR1 upregulation mediates tolerance after brain ischemic preconditioning

    Journal of Cerebral Blood Flow and Metabolism, Vol. 25, Núm. 2, pp. 193-203

  4. The nonthiazolidinedione PPARγ agonist L-796,449 is neuroprotective in experimental stroke

    Journal of Neuropathology and Experimental Neurology, Vol. 64, Núm. 9, pp. 797-805

2004

  1. Brain oxidative markers in stress: Posible new drug targets against neuroinflammation

    Current Neuropharmacology, Vol. 2, Núm. 2, pp. 183-189

  2. In Vitro Ischemic Tolerance Involves Upregulation of Glutamate Transport Partly Mediated by the TACE/ADAM17-Tumor Necrosis Factor-α Pathway

    Journal of Neuroscience, Vol. 24, Núm. 6, pp. 1350-1357

2003

  1. Expression and function of tumour necrosis factor-α-converting enzyme in the central nervous system

    NeuroSignals, Vol. 12, Núm. 2, pp. 53-58

  2. Inhibition of glutamate release by delaying ATP fall accounts for neuroprotective effects of antioxidants in experimental stroke.

    The FASEB journal : official publication of the Federation of American Societies for Experimental Biology, Vol. 17, Núm. 14, pp. 2082-2084

2002

  1. TACE/ADAM17-TNF-α pathway in rat cortical cultures after exposure to oxygen-glucose deprivation or glutamate

    Journal of Cerebral Blood Flow and Metabolism, Vol. 22, Núm. 5, pp. 576-585

  2. The increase in TNF-α levels is implicated in NF-κB activation and inducible nitric oxide synthase expression in brain cortex after immobilization stress

    Neuropsychopharmacology, Vol. 26, Núm. 2, pp. 155-163

2001

  1. Implication of TNF-α convertase (TACE/ADAM17) in inducible nitric oxide synthase expression and inflammation in an experimental model of colitis

    Cytokine, Vol. 16, Núm. 6, pp. 220-226

  2. Up-regulation of TNF-α convertase (TACE/ADAM17) after oxygen-glucose deprivation in rat forebrain slices

    Neuropharmacology, Vol. 40, Núm. 8, pp. 1094-1102

2000

  1. Fructose-1,6-bisphosphate inhibits the expression of inducible nitric oxide synthase caused by oxygen-glucose deprivation through the inhibition of glutamate release in rat forebrain slices

    Naunyn-Schmiedeberg's Archives of Pharmacology, Vol. 362, Núm. 3, pp. 208-212