ASUNCIÓN
LÓPEZ-CALDERÓN BARREDA
Catedrática de universidad
MARÍA ÁNGELES
VILLANÚA BERNUÉS
Chercheuse jusqu' 2020
Publications dans lesquelles il/elle collabore avec MARÍA ÁNGELES VILLANÚA BERNUÉS (39)
2016
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D-TRP(8)-γMSH Prevents the Effects of Endotoxin in Rat Skeletal Muscle Cells through TNFα/NF-KB Signalling Pathway
PLoS ONE, Vol. 11, Núm. 5
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Formoterol decreases muscle wasting as well as inflammation in the rat model of rheumatoid arthritis
American Journal of Physiology - Endocrinology and Metabolism, Vol. 310, Núm. 11, pp. E925-E937
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The melanocortin receptor type 3 agonist d-Trp(8)-γMSH decreases inflammation and muscle wasting in arthritic rats
Journal of Cachexia, Sarcopenia and Muscle, Vol. 7, Núm. 1, pp. 79-89
2014
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α MSH Blunts Endotoxin-Induced MuRF1 and Atrogin-1 Upregulation in Skeletal Muscle by Modulating NF- B and Akt/FoxO1 Pathway
Mediators of Inflammation, Vol. 2014
2013
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Systemic α-melanocyte-stimulating hormone administration decreases arthritis-induced anorexia and muscle wasting
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, Vol. 304, Núm. 10
2012
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Effect of cyclooxygenase-2 inhibition by meloxicam, on atrogin-1 and myogenic regulatory factors in skeletal muscle of rats injected with endotoxin
Journal of Physiology and Pharmacology, Vol. 63, Núm. 6, pp. 649-659
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Short-term growth hormone or IGF-I administration improves the IGF-IGFBP system in arthritic rats
Growth Hormone and IGF Research, Vol. 22, Núm. 1, pp. 22-29
2011
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Comparison of the effects of the n-3 polyunsaturated fatty acid eicosapentaenoic and fenofibrate on the inhibitory effect of arthritis on IGF1
Journal of Endocrinology, Vol. 210, Núm. 3, pp. 361-368
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Fenofibrate, a PPARα agonist, decreases atrogenes and myostatin expression and improves arthritis-induced skeletal muscle atrophy
American Journal of Physiology - Endocrinology and Metabolism, Vol. 300, Núm. 5
2010
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Systemic IGF-I administration attenuates the inhibitory effect of chronic arthritis on gastrocnemius mass and decreases atrogin-1 and IGFBP-3
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, Vol. 299, Núm. 2
2009
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Cyclooxygenase-2 inhibition reverts the decrease in adiponectin levels and attenuates the loss of white adipose tissue during chronic inflammation
European Journal of Pharmacology, Vol. 608, Núm. 1-3, pp. 97-103
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Eicosapentaenoic acid attenuates arthritis-induced muscle wasting acting on atrogin-1 and on myogenic regulatory factors
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, Vol. 297, Núm. 5
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IGF-I system, atrogenes and myogenic regulatory factors in arthritis induced muscle wasting
Molecular and Cellular Endocrinology, Vol. 309, Núm. 1-2, pp. 8-16
2008
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Adipose tissue loss in adjuvant arthritis is associated with a decrease in lipogenesis, but not with an increase in lipolysis
Journal of Endocrinology, Vol. 197, Núm. 1, pp. 111-119
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GH-releasing peptide-2 administration prevents liver inflammatory response in endotoxemia
American Journal of Physiology - Endocrinology and Metabolism, Vol. 294, Núm. 1
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Ptgs2 activation by endotoxin mediates the decrease in Igf1, but not in Igfbp3, gene expression in the liver
Journal of Endocrinology, Vol. 198, Núm. 2, pp. 385-394
2007
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Experimental arthritis inhibits the insulin-like growth factor-I axis and induces muscle wasting through cyclooxygenase-2 activation
American Journal of Physiology - Endocrinology and Metabolism, Vol. 292, Núm. 6
2006
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Inactivation of Kupffer cells by gadolinium administration prevents lipopolysaccharide-induced decrease in liver insulin-like growth factor-I and IGF-binding protein-3 gene expression
Journal of Endocrinology, Vol. 188, Núm. 3, pp. 503-511
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Nitric oxide production by hepatocytes contributes to the inhibitory effect of endotoxin on insulin-like growth factor I gene expression
Journal of Endocrinology, Vol. 190, Núm. 3, pp. 847-856
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Tumour necrosis factor blockade did not prevent the increase of muscular muscle RING finger-1 and muscle atrophy F-box in arthritis rats
Journal of Endocrinology, Vol. 191, Núm. 1, pp. 319-326