Publicaciones en colaboración con investigadores/as de Instituto Cajal (10)

2014

  1. The JNK inhibitor, SP600125, potentiates the glial response and cell death induced by methamphetamine in the mouse striatum

    International Journal of Neuropsychopharmacology, Vol. 17, Núm. 2, pp. 235-246

2013

  1. Cocaine potentiates MDMA-induced oxidative stress but not dopaminergic neurotoxicity in mice: Implications for the pathogenesis of free radical-induced neurodegenerative disorders

    Psychopharmacology, Vol. 230, Núm. 1, pp. 125-135

2012

  1. Dopamine D 1 receptor deletion strongly reduces neurotoxic effects of methamphetamine

    Neurobiology of Disease, Vol. 45, Núm. 2, pp. 810-820

2011

  1. Dopamine D2-receptor knockout mice are protected against dopaminergic neurotoxicity induced by methamphetamine or MDMA

    Neurobiology of Disease, Vol. 42, Núm. 3, pp. 391-403

2010

  1. Selective vulnerability in striosomes and in the nigrostriatal dopaminergic pathway after methamphetamine administration : early loss of TH in striosomes after methamphetamine.

    Neurotoxicity research, Vol. 18, Núm. 1, pp. 48-58

2008

  1. Early loss of dopaminergic terminals in striosomes after MDMA administration to mice

    Synapse, Vol. 62, Núm. 1, pp. 80-84

  2. Persistent MDMA-induced dopaminergic neurotoxicity in the striatum and substantia nigra of mice

    Journal of Neurochemistry, Vol. 107, Núm. 4, pp. 1102-1112

1994

  1. Neonatal guanethidine sympathectomy suppresses autotomy and prevents changes in spinal and supraspinal monoamine levels induced by peripheral deafferentation in rats

    Pain, Vol. 56, Núm. 1, pp. 3-8

1993

  1. Molecular mechanisms of pain: Serotonin1A receptor agonists trigger transactivation by c-fos of the prodynorphin gene in spinal cord neurons

    Neuron, Vol. 10, Núm. 4, pp. 599-611

1988

  1. Unilateral dorsal rhizotomy decreases monoamine levels in the rat spinal cord

    Neuroscience Letters, Vol. 87, Núm. 3, pp. 302-306