Publications dans lesquelles il/elle collabore avec JOSÉ SÁNCHEZ-PRIETO BORJA (16)

2023

  1. Control of a hippocampal recurrent excitatory circuit by cannabinoid receptor-interacting protein Gap43

    Nature Communications, Vol. 14, Núm. 1

  2. The activation of mGluR4 rescues parallel fiber synaptic transmission and LTP, motor learning and social behavior in a mouse model of Fragile X Syndrome

    Molecular autism, Vol. 14, Núm. 1, pp. 14

2021

  1. Identification of bip as a cb1 receptor-interacting protein that fine-tunes cannabinoid signaling in the mouse brain

    Journal of Neuroscience, Vol. 41, Núm. 38, pp. 7924-7941

2020

  1. Endocannabinoid signalling in stem cells and cerebral organoids drives differentiation to deep layer projection neurons via CB1 receptors

    Development (Cambridge, England), Vol. 147, Núm. 24

  2. β-adrenergic receptors/epac signaling increases the size of the readily releasable pool of synaptic vesicles required for parallel fiber LTP

    Journal of Neuroscience, Vol. 40, Núm. 45, pp. 8604-8617

2019

  1. The loss of β adrenergic receptor mediated release potentiation in a mouse model of fragile X syndrome

    Neurobiology of Disease, Vol. 130

2018

  1. AhR deletion promotes aberrant morphogenesis and synaptic activity of adult-generated granule neurons and impairs hippocampus-dependent memory

    eNeuro, Vol. 5, Núm. 4

  2. Bidirectional modulation of glutamatergic synaptic transmission by metabotropic glutamate type 7 receptors at Schaffer collateral–CA1 hippocampal synapses

    Journal of Physiology, Vol. 596, Núm. 5, pp. 921-940

2016

  1. Cross-talk between metabotropic glutamate receptor 7 and beta adrenergic receptor signaling at cerebrocortical nerve terminals

    Neuropharmacology, Vol. 101, pp. 412-425

2011

  1. Non-additive potentiation of glutamate release by phorbol esters and metabotropic mGlu7 receptor in cerebrocortical nerve terminals

    Journal of Neurochemistry, Vol. 116, Núm. 4, pp. 476-485

2010

  1. The metabotropic glutamate receptor mGlu7 activates phospholipase C, translocates munc-13-1 protein, and potentiates glutamate release at cerebrocortical nerve terminals

    Journal of Biological Chemistry, Vol. 285, Núm. 23, pp. 17907-17917

2009

  1. Partial compensation for N-type Ca2+ channel loss by P/Q-type Ca2+ channels underlines the differential release properties supported by these channels at cerebrocortical nerve terminals

    European Journal of Neuroscience, Vol. 29, Núm. 6, pp. 1131-1140

  2. Subcellular compartment-specific molecular diversity of pre- and post-synaptic GABAB-activated GIRK channels in Purkinje cells

    Journal of Neurochemistry, Vol. 110, Núm. 4, pp. 1363-1376

2008

  1. The inhibition of release by mGlu7 receptors is independent of the Ca2+ channel type but associated to GABAB and adenosine A1 receptors

    Neuropharmacology, Vol. 55, Núm. 4, pp. 464-473

2007

  1. The coexistence of multiple receptors in a single nerve terminal provides evidence for pre-synaptic integration

    Journal of Neurochemistry, Vol. 103, Núm. 6, pp. 2314-2326

  2. mGluR7 inhibits glutamate release through a PKC-independent decrease in the activity of P/Q-type Ca2+ channels and by diminishing cAMP in hippocampal nerve terminals

    European Journal of Neuroscience, Vol. 26, Núm. 2, pp. 312-322