MARÍA TERESA
FREJO MOYA
Profesora titular de universidad
PAULA VIVIANA
MOYANO-CIRES IVANOFF
Profesora ayudante doctora
Publications by the researcher in collaboration with PAULA VIVIANA MOYANO-CIRES IVANOFF (21)
2022
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Cadmium-induced neurotoxic effects on rat basal forebrain cholinergic system through thyroid hormones disruption
Environmental Toxicology and Pharmacology, Vol. 90
2021
2020
2019
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Manganese induced ROS and AChE variants alteration leads to SN56 basal forebrain cholinergic neuronal loss after acute and long-term treatment
Food and Chemical Toxicology, Vol. 125, pp. 583-594
2018
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Cadmium alters heat shock protein pathways in SN56 cholinergic neurons, leading to Aβ and phosphorylated Tau protein generation and cell death
Food and Chemical Toxicology, Vol. 121, pp. 297-308
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Cadmium induced ROS alters M1 and M3 receptors, leading to SN56 cholinergic neuronal loss, through AChE variants disruption
Toxicology, Vol. 394, pp. 54-62
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SN56 neuronal cell death after 24 h and 14 days chlorpyrifos exposure through glutamate transmission dysfunction, increase of GSK-3β enzyme, β-amyloid and tau protein levels
Toxicology, Vol. 402-403, pp. 17-27
2017
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Amitraz changes NE, DA and 5-HT biosynthesis and metabolism mediated by alterations in estradiol content in CNS of male rats
Chemosphere, Vol. 181, pp. 518-529
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Effects of sex hormones disruption, after prenatal and postnatal exposure to chlordimeform, on monoaminergic neurotransmitters systems in female and male rat’s prefrontal cortex
Medicina balear, Vol. 32, Núm. 1, pp. 11-19
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Hippocampal alteration of monoaminergic neurotransmitters biosynthesis and metabolism in CNS in rats, after prenatal and postnatal exposure to chlordimeform, through sex hormones disruption
Medicina balear, Vol. 32, Núm. 3, pp. 31-39
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Primary hippocampal neuronal cell death induction after acute and repeated paraquat exposures mediated by AChE variants alteration and cholinergic and glutamatergic transmission disruption
Toxicology, Vol. 390, pp. 88-99
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Striatal alteration of monoaminergic neurotransmitters systems in rats, after prenatal and postnatal exposure to chlordimeform, through testosterone and estradiol disruption
Medicina balear, Vol. 32, Núm. 2, pp. 13-22
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Toxicogenomic profile of apoptotic and necrotic SN56 basal forebrain cholinergic neuronal loss after acute and long-term chlorpyrifos exposure
Neurotoxicology and Teratology, Vol. 59, pp. 68-73
2016
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Apoptotic and necrotic basal forebrain cholinergic neuronal loss and dendritic spines alteration after acute and long-term chlorpyrifos exposure: Legal implications of the use of toxicogenomic profile as a biomarker of harmful effects induced under subclinical doses
Medicina balear, Vol. 31, Núm. 1, pp. 24-34
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Cadmium-induced cell death of basal forebrain cholinergic neurons mediated by muscarinic M1 receptor blockade, increase in GSK-3β enzyme, β-amyloid and tau protein levels
Archives of Toxicology, Vol. 90, Núm. 5, pp. 1081-1092
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Muscarinic M1 receptor partially modulates higher sensitivity to cadmium-induced cell death in primary basal forebrain cholinergic neurons: A cholinesterase variants dependent mechanism
Toxicology, Vol. 361-362, pp. 1-11
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SN56 basal forebrain cholinergic neuronal loss after acute and long-term chlorpyrifos exposure through oxidative stress generation; P75NTR and α7-nAChRs alterations mediated partially by AChE variants disruption
Toxicology, Vol. 353-354, pp. 48-57
2015
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Acute and long-term exposure to chlorpyrifos induces cell death of basal forebrain cholinergic neurons through AChE variants alteration
Toxicology, Vol. 336, pp. 1-9
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Impaired glutamatergic and GABAergic transmission by amitraz in primary hippocampal cells
Neurotoxicology and Teratology, Vol. 50, pp. 82-87
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Molecular Mechanisms of Amitraz Mammalian Toxicity: A Comprehensive Review of Existing Data
Chemical Research in Toxicology, Vol. 28, Núm. 6, pp. 1073-1094