LISARDO
BOSCA GOMAR
Profesor asociado
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Universitat de Barcelona
Barcelona, EspañaPublicaciones en colaboración con investigadores/as de Universitat de Barcelona (11)
2024
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Endogenous LXR signaling controls pulmonary surfactant homeostasis and prevents lung inflammation
Cellular and Molecular Life Sciences, Vol. 81, Núm. 1
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Trabectedin and Lurbinectedin Modulate the Interplay between Cells in the Tumour Microenvironment—Progresses in Their Use in Combined Cancer Therapy
Molecules, Vol. 29, Núm. 2
2023
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Defining the metabolic signatures associated with human macrophage polarisation
Biochemical Society transactions, Vol. 51, Núm. 4, pp. 1429-1436
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Immunometabolic actions of trabectedin and lurbinectedin on human macrophages: relevance for their anti-tumor activity
Frontiers in Immunology, Vol. 14
2021
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Crosstalk Between LXR and Caveolin-1 Signaling Supports Cholesterol Efflux and Anti-Inflammatory Pathways in Macrophages
Frontiers in Endocrinology, Vol. 12
2015
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Epigenetics override pro-inflammatory PTGS transcriptomic signature towards selective hyperactivation of PGE2 in colorectal cancer
Clinical Epigenetics, Vol. 7, Núm. 1
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HIF-1α and PFKFB3 mediate a tight relationship between proinflammatory activation and anerobic metabolism in atherosclerotic macrophages
Arteriosclerosis, Thrombosis, and Vascular Biology, Vol. 35, Núm. 6, pp. 1463-1471
2012
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Relevance of the MEK/ERK signaling pathway in the metabolism of activated macrophages: A metabolomic approach
Journal of Immunology, Vol. 188, Núm. 3, pp. 1402-1410
2010
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Substrate fate in activated macrophages: A comparison between innate, classic, and alternative activation
Journal of Immunology, Vol. 185, Núm. 1, pp. 605-614
2005
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Assessment of a dual regulatory role for NO in liver regeneration after partial hepatectomy: Protection against apoptosis and retardation of hepatocyte proliferation
FASEB Journal, Vol. 19, Núm. 8, pp. 995-997
1997
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Macrophage Triggering with Cecropin A and Melittin-Derived Peptides Induces Type II Nitric Oxide Synthase Expression
Journal of Immunology, Vol. 158, Núm. 9, pp. 4437-4443