LISARDO
BOSCA GOMAR
Profesor asociado
Hospital Universitario La Paz
Madrid, EspañaPublicaciones en colaboración con investigadores/as de Hospital Universitario La Paz (24)
2022
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Specialized Proresolving Mediators Protect Against Experimental Autoimmune Myocarditis by Modulating Ca2+ Handling and NRF2 Activation
JACC: Basic to Translational Science, Vol. 7, Núm. 6, pp. 544-560
2021
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Resolution-Based Therapies: The Potential of Lipoxins to Treat Human Diseases
Frontiers in Immunology, Vol. 12
2020
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BML-111 treatment prevents cardiac apoptosis and oxidative stress in a mouse model of autoimmune myocarditis
FASEB Journal, Vol. 34, Núm. 8, pp. 10531-10546
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Innate Immune Receptors, Key Actors in Cardiovascular Diseases
JACC: Basic to Translational Science, Vol. 5, Núm. 7, pp. 735-749
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Specific effects of trabectedin and lurbinectedin on human macrophage function and fate—novel insights
Cancers, Vol. 12, Núm. 10, pp. 1-22
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Tumor stem cells fuse with monocytes to form highly invasive tumor-hybrid cells
OncoImmunology, Vol. 9, Núm. 1
2019
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Protective Role of Hepatocyte Cyclooxygenase-2 Expression Against Liver Ischemia–Reperfusion Injury in Mice
Hepatology, Vol. 70, Núm. 2, pp. 650-665
2018
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Deficiency of NOD1 improves the β-adrenergic modulation of Ca2+ handling in a mouse model of heart failure
Frontiers in Physiology, Vol. 9, Núm. JUN
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PGE2 induces apoptosis of hepatic stellate cells and attenuates liver fibrosis in mice by downregulating miR-23a-5p and miR-28a-5p
Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1864, Núm. 2, pp. 325-337
2017
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NOD1 activation in cardiac fibroblasts induces myocardial fibrosis in a murine model of type 2 diabetes
Biochemical Journal, Vol. 474, Núm. 3, pp. 399-410
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Role of NOD1 in Heart Failure Progression via Regulation of Ca2+ Handling
Journal of the American College of Cardiology, Vol. 69, Núm. 4, pp. 423-433
2016
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Cell expansion-dependent inflammatory and metabolic profile of human bone marrow mesenchymal stem cells
Frontiers in Physiology, Vol. 7, Núm. NOV
2015
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A labdane diterpene exerts ex vivo and in vivo cardioprotection against post-ischemic injury: Involvement of AKT-dependent mechanisms
Biochemical Pharmacology, Vol. 93, Núm. 4, pp. 428-439
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HIF-1α and PFKFB3 mediate a tight relationship between proinflammatory activation and anerobic metabolism in atherosclerotic macrophages
Arteriosclerosis, Thrombosis, and Vascular Biology, Vol. 35, Núm. 6, pp. 1463-1471
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NOD1, a new player in cardiac function and calcium handling
Cardiovascular Research, Vol. 106, Núm. 3, pp. 375-386
2014
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Involvement of monocytes/macrophages as key factors in the development and progression of cardiovascular diseases
Biochemical Journal, Vol. 458, Núm. 2, pp. 187-193
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Mitochondrial DAMPs induce endotoxin tolerance in human monocytes: An observation in patients with myocardial infarction
PLoS ONE, Vol. 9, Núm. 5
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NFκB2/p100 is a key factor for endotoxin tolerance in human monocytes: A demonstration using primary human monocytes from patients with sepsis
Journal of Immunology, Vol. 193, Núm. 8, pp. 4195-4202
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NOD1 receptor is up-regulated in diabetic human and murine myocardium
Clinical Science, Vol. 127, Núm. 12, pp. 665-677
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Prolonged leptin treatment increases transient outward K+ current via upregulation of Kv4.2 and Kv4.3 channel subunits in adult rat ventricular myocytes
Pflugers Archiv European Journal of Physiology, Vol. 466, Núm. 5, pp. 903-914