Publikationen in Zusammenarbeit mit Forschern von Icahn School of Medicine at Mount Sinai (13)

2022

  1. Activation of the unfolded protein response (Upr) is associated with cholangiocellular injury, fibrosis and carcinogenesis in an experimental model of fibropolycystic liver disease

    Cancers, Vol. 14, Núm. 1

2021

  1. Anti-TNFα treatment in Crohn’s disease: Impact on hepatic steatosis, gut-derived hormones and metabolic status

    Liver International, Vol. 41, Núm. 11, pp. 2646-2658

2020

  1. Altered Microbiota Diversity and Bile Acid Signaling in Cirrhotic and Noncirrhotic NASH-HCC

    Clinical and translational gastroenterology, Vol. 11, Núm. 3, pp. e00131

  2. Hepatocyte KLF6 expression affects FXR signalling and the clinical course of primary sclerosing cholangitis

    Liver International, Vol. 40, Núm. 9, pp. 2172-2181

2017

  1. Krüppel-like factor 6 is a transcriptional activator of autophagy in acute liver injury

    Scientific Reports, Vol. 7, Núm. 1

2014

  1. Extracellular matrix and liver disease

    Antioxidants and Redox Signaling, Vol. 21, Núm. 7, pp. 1078-1097

2012

  1. Arachidonic acid stimulates TNFα production in Kupffer cells via a reactive oxygen species-pERK1/2-Egr1-dependent mechanism

    American Journal of Physiology - Gastrointestinal and Liver Physiology, Vol. 303, Núm. 2

  2. Oxidative Stress Modulates KLF6Full and Its Splice Variants

    Alcoholism: Clinical and Experimental Research, Vol. 36, Núm. 11, pp. 1851-1862

2011

  1. Mechanisms of liver fibrosis associated with experimental fasciola hepatica infection: Roles of Fas2 proteinase and hepatic stellate cell activation

    Journal of Parasitology, Vol. 97, Núm. 1, pp. 82-87

2009

  1. Alcohol and liver fibrosis

    Seminars in Liver Disease, Vol. 29, Núm. 2, pp. 211-221

  2. Reactive Nitrogen Species Switch on Early Extracellular Matrix Remodeling via Induction of MMP1 and TNFα

    Gastroenterology, Vol. 136, Núm. 4

2008

  1. Ethanol and arachidonic acid synergize to activate Kupffer cells and modulate the fibrogenic response via tumor necrosis factor α, reduced glutathione, and transforming growth factor β-dependent mechanisms

    Hepatology, Vol. 48, Núm. 6, pp. 2027-2039

2006

  1. Células de Kupffer y hepatopatía alcohólica

    Revista Espanola de Enfermedades Digestivas, Vol. 98, Núm. 6, pp. 460-472