Publicaciones en las que colabora con JOSÉ MARÍA LADERO QUESADA (40)

2012

  1. Polymorphism of the TLR4 gene reduces the risk of hepatitis C virus-induced hepatocellular carcinoma

    Oncology, Vol. 82, Núm. 1, pp. 35-40

  2. Predicting response to therapy in chronic hepatitis C: An approach combining interleukin-28B gene polymorphisms and clinical data

    Journal of Gastroenterology and Hepatology (Australia), Vol. 27, Núm. 2, pp. 279-285

  3. Relation of IL28b gene polymorphism with biochemical and histological features in hepatitis C virus-induced liver disease

    PLoS ONE, Vol. 7, Núm. 5

2010

  1. Non-invasive evaluation of the fibrosis stage in chronic hepatitis C: A comparative analysis of nine scoring methods

    Scandinavian Journal of Gastroenterology, Vol. 45, Núm. 1, pp. 51-59

2009

  1. Analysis of a non-synonymous single nucleotide polymorphism of the human diamine oxidase gene (ref. SNP ID: Rs1049793) in patients with Crohn's disease

    Scandinavian Journal of Gastroenterology, Vol. 44, Núm. 10, pp. 1207-1212

2008

  1. "12 Weeks' stopping rule" in the treatment of genotype 1 chronic hepatitis C: Two prognostic categories under the same label?

    Scandinavian Journal of Gastroenterology, Vol. 43, Núm. 8, pp. 979-983

  2. Interaction of CYP2C8 and CYP2C9 genotypes modifies the risk for nonsteroidal anti-inflammatory drugs-related acute gastrointestinal bleeding

    Pharmacogenetics and Genomics, Vol. 18, Núm. 1, pp. 37-43

2007

  1. GSTT1 and GSTM1 null genotypes may facilitate hepatitis C virus infection becoming chronic

    Journal of Infectious Diseases, Vol. 195, Núm. 9, pp. 1320-1323

  2. Genetic variability in CYP3A4 and CYP3A5 in primary liver, gastric and colorectal cancer patients

    BMC Cancer, Vol. 7

  3. Glutathione S-transferases π1, α1 and μ3 genetic polymorphisms and the risk of hepatocellular carcinoma in humans

    Pharmacogenomics, Vol. 8, Núm. 8, pp. 895-899

  4. Utilidad de la biopsia hepática en el diagnóstico etiológico de las alteraciones de la analítica hepática de causa incierta

    Gastroenterologia y Hepatologia, Vol. 30, Núm. 6, pp. 325-330

2006

  1. Glutathione S-transferase M1 and T1 genetic polymorphisms are not related to the risk of hepatocellular carcinoma: A study in the Spanish population

    European Journal of Cancer, Vol. 42, Núm. 1, pp. 73-77

  2. Glutathione S-transferases μ1, θ1, π1 α1 and μ3 genetic polymorphisms and the risk of colorectal and gastric cancers in humans

    Pharmacogenomics, Vol. 7, Núm. 5, pp. 711-718

  3. Severity of ulcerative colitis is associated with a polymorphism at diamine oxidase gene but not at histamine N-methyltransferase gene

    World Journal of Gastroenterology, Vol. 12, Núm. 4, pp. 615-620

2005

  1. A computer-assisted morphometric quantitative analysis of iron overload in liver biopsies. A comparison with histological and biochemical methods

    Pathology Research and Practice, Vol. 201, Núm. 10, pp. 673-677

  2. Polymorhisms in the transforming growth factor-β1 gene (TGF-β1) and the risk of advanced alcoholic liver disease

    Liver International, Vol. 25, Núm. 5, pp. 935-939

  3. Polymorphisms of the glutathione S-transferases mu-1 (GSTM1) and theta-1 (GSTT1) and the risk of advanced alcoholic liver disease

    Scandinavian Journal of Gastroenterology, Vol. 40, Núm. 3, pp. 348-353

2004

  1. Genetic predisposition to acute gastrointestinal bleeding after NSAIDs use

    British Journal of Pharmacology, Vol. 141, Núm. 2, pp. 205-208

  2. Leflunomide-induced acute hepatitis

    Digestive and Liver Disease, Vol. 36, Núm. 1, pp. 82-84

  3. Síndrome hereditario de hiperferritinemia y cataratas. Descripción de una nueva familia en España

    Revista Espanola de Enfermedades Digestivas, Vol. 96, Núm. 7, pp. 507-511