MARGARITA
FERNÁNDEZ GARCÍA DE CASTRO
Forscherin bis um 2017
MARÍA ISABEL
FABREGAT ROMERO
Profesora titular de universidad
Publikationen, an denen er mitarbeitet MARÍA ISABEL FABREGAT ROMERO (24)
2018
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Bone morphogenetic protein 9 as a key regulator of liver progenitor cells in DDC-induced cholestatic liver injury
Liver International, Vol. 38, Núm. 9, pp. 1664-1675
2016
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Dissecting the role of epidermal growth factor receptor catalytic activity during liver regeneration and hepatocarcinogenesis
Hepatology, Vol. 63, Núm. 2, pp. 604-619
2015
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BMP9-induced survival effect in liver tumor cells requires p38MAPK activation
International Journal of Molecular Sciences, Vol. 16, Núm. 9, pp. 20431-20448
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HGF/c-Met signaling promotes liver progenitor cell migration and invasion by an epithelial-mesenchymal transition-independent, phosphatidyl inositol-3 kinase-dependent pathway in an in vitro model
Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1853, Núm. 10, pp. 2453-2463
2014
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Polμ deficiency increases resistance to oxidative damage and delays liver aging
PLoS ONE, Vol. 9, Núm. 4
2013
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BMP9 Is a Proliferative and Survival Factor for Human Hepatocellular Carcinoma Cells
PLoS ONE, Vol. 8, Núm. 7
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Mouse Hepatic Oval Cells Require Met-Dependent PI3K to Impair TGF-β-Induced Oxidative Stress and Apoptosis
PLoS ONE, Vol. 8, Núm. 1
2012
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EGFR is dispensable for c-Met-mediated proliferation and survival activities in mouse adult liver oval cells
Cellular Signalling, Vol. 24, Núm. 2, pp. 505-513
2008
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Deletion of the Met tyrosine kinase in liver progenitor oval cells increases sensitivity to apoptosis in vitro
American Journal of Pathology, Vol. 172, Núm. 5, pp. 1238-1247
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Isolation and characterization of a putative liver progenitor population after treatment of fetal rat hepatocytes with TGF-beta
Journal of Cellular Physiology, Vol. 215, Núm. 3, pp. 846-855
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Upregulation of the NADPH oxidase NOX4 by TGF-beta in hepatocytes is required for its pro-apoptotic activity
Journal of Hepatology, Vol. 49, Núm. 6, pp. 965-976
2007
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Activation of NADPH oxidase by transforming growth factor-β in hepatocytes mediates up-regulation of epidermal growth factor receptor ligands through a nuclear factor-κB-dependent mechanism
Biochemical Journal, Vol. 405, Núm. 2, pp. 251-259
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Survival and apoptosis: A dysregulated balance in liver cancer
Liver International, Vol. 27, Núm. 2, pp. 155-162
2006
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Autocrine production of TGF-β confers resistance to apoptosis after an epithelial-mesenchymal transition process in hepatocytes: Role of EGF receptor ligands
Experimental Cell Research, Vol. 312, Núm. 15, pp. 2860-2871
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EGF blocks NADPH oxidase activation by TGF-β in fetal rat hepatocytes, impairing oxidative stress, and cell death
Journal of Cellular Physiology, Vol. 207, Núm. 2, pp. 322-330
2004
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Resistance to TGF-β-induced apoptosis in regenerating hepatocytes
Journal of Cellular Physiology, Vol. 201, Núm. 3, pp. 385-392
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Source of early reactive oxygen species in the apoptosis induced by transforming growth factor-β in fetal rat hepatocytes
Free Radical Biology and Medicine, Vol. 36, Núm. 1, pp. 16-26
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Transforming growth factor-beta activates both pro-apoptotic and survival signals in fetal rat hepatocytes
Experimental Cell Research, Vol. 292, Núm. 1, pp. 209-218
2002
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The epithelial mesenchymal transition confers resistance to the apoptotic effects of transforming growth factor β in fetal rat hepatocytes
Molecular Cancer Research, Vol. 1, Núm. 1, pp. 68-78
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cIAP-1, but not XIAP, is cleaved by caspases during the apoptosis induced by TGF-β in fetal rat hepatocytes
FEBS Letters, Vol. 520, Núm. 1-3, pp. 93-96