César Roncero Maíllo-rekin lankidetzan egindako argitalpenak (18)

2018

  1. Bone morphogenetic protein 9 as a key regulator of liver progenitor cells in DDC-induced cholestatic liver injury

    Liver International, Vol. 38, Núm. 9, pp. 1664-1675

2016

  1. Dissecting the role of epidermal growth factor receptor catalytic activity during liver regeneration and hepatocarcinogenesis

    Hepatology, Vol. 63, Núm. 2, pp. 604-619

2015

  1. BMP9-induced survival effect in liver tumor cells requires p38MAPK activation

    International Journal of Molecular Sciences, Vol. 16, Núm. 9, pp. 20431-20448

  2. HGF/c-Met signaling promotes liver progenitor cell migration and invasion by an epithelial-mesenchymal transition-independent, phosphatidyl inositol-3 kinase-dependent pathway in an in vitro model

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1853, Núm. 10, pp. 2453-2463

2013

  1. BMP9 Is a Proliferative and Survival Factor for Human Hepatocellular Carcinoma Cells

    PLoS ONE, Vol. 8, Núm. 7

  2. Mouse Hepatic Oval Cells Require Met-Dependent PI3K to Impair TGF-β-Induced Oxidative Stress and Apoptosis

    PLoS ONE, Vol. 8, Núm. 1

2012

  1. EGFR is dispensable for c-Met-mediated proliferation and survival activities in mouse adult liver oval cells

    Cellular Signalling, Vol. 24, Núm. 2, pp. 505-513

2008

  1. Upregulation of the NADPH oxidase NOX4 by TGF-beta in hepatocytes is required for its pro-apoptotic activity

    Journal of Hepatology, Vol. 49, Núm. 6, pp. 965-976

2007

  1. Activation of NADPH oxidase by transforming growth factor-β in hepatocytes mediates up-regulation of epidermal growth factor receptor ligands through a nuclear factor-κB-dependent mechanism

    Biochemical Journal, Vol. 405, Núm. 2, pp. 251-259

  2. Survival and apoptosis: A dysregulated balance in liver cancer

    Liver International, Vol. 27, Núm. 2, pp. 155-162

2006

  1. EGF blocks NADPH oxidase activation by TGF-β in fetal rat hepatocytes, impairing oxidative stress, and cell death

    Journal of Cellular Physiology, Vol. 207, Núm. 2, pp. 322-330

2001

  1. Activation of caspases occurs downstream from radical oxygen species production, Bcl-xL down-regulation, and early cytochrome c release in apoptosis induced by transforming growth factor β in rat fetal hepatocytes

    Hepatology, Vol. 34, Núm. 3, pp. 548-556

  2. Activation of p38MAPK by TGF-β in fetal rat hepatocytes requires radical oxygen production, but is dispensable for cell death

    FEBS Letters, Vol. 499, Núm. 3, pp. 225-229

  3. Reactive oxygen species (ROS) mediates the mitochondrial-dependent apoptosis induced by transforming growth factor β in fetal hepatocytes

    FASEB Journal, Vol. 15, Núm. 3, pp. 741-751

2000

  1. Epidermal growth factor impairs the cytochrome C/caspase-3 apoptotic pathway induced by transforming growth factor β in rat fetal hepatocytes via a phosphoinositide 3-kinase-dependent pathway

    Hepatology, Vol. 32, Núm. 3, pp. 528-535

1996

  1. Triiodothyronine induces the transcription of the uncoupling protein gene and stabilizes its mRNA in fetal rat brown adipocyte primary cultures

    Journal of Biological Chemistry, Vol. 271, Núm. 4, pp. 2076-2081

1994

  1. Triiodothyronine induces the expression of the uncoupling protein in long term fetal rat brown adipocyte primary cultures: Role of nuclear thyroid hormone receptor expression

    Endocrinology, Vol. 134, Núm. 3, pp. 1067-1074

1984

  1. Preparation of hormone-sensitive adipocytes from foetal rat brown adipose tissue

    Bioscience Reports, Vol. 4, Núm. 5, pp. 397-401