Publicaciones en las que colabora con José Manuel Cuezva Marcos (11)

2010

  1. Human G3BP1 interacts with β-F1-ATPase mRNA and inhibits its translation

    Journal of Cell Science, Vol. 123, Núm. 16, pp. 2685-2696

  2. Selective inhibition of β-F1-ATPase mRNA translation in human tumours

    Biochemical Journal, Vol. 426, Núm. 3, pp. 319-326

  3. Up-regulation of the ATPase Inhibitory Factor 1 (IF1) of the mitochondrial H+-ATP synthase in human tumors mediates the metabolic shift of cancer cells to a warburg phenotype

    Journal of Biological Chemistry, Vol. 285, Núm. 33, pp. 25308-25313

2009

  1. Cancer abolishes the tissue type-specific differences in the phenotype of energetic metabolism

    Translational Oncology, Vol. 2, Núm. 3, pp. 138-145

  2. Glucose avidity of carcinomas

    Cancer Letters, Vol. 276, Núm. 2, pp. 125-135

  3. The tumor suppressor function of mitochondria: Translation into the clinics

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1792, Núm. 12, pp. 1145-1158

2008

  1. HuR and the bioenergetic signature of breast cancer: A low tumor expression of the RNA-binding protein predicts a higher risk of disease recurrence

    Carcinogenesis, Vol. 29, Núm. 11, pp. 2053-2061

2007

  1. A message emerging from development: The repression of mitochondrial β-F1-ATPase expression in cancer

    Journal of Bioenergetics and Biomembranes

  2. Loss of the mitochondrial bioenergetic capacity underlies the glucose avidity of carcinomas

    Cancer Research, Vol. 67, Núm. 19, pp. 9013-9017

2006

  1. Biogenesis and dynamic of mitochondria during the cell cycle: Significance of 3'UTRs

    PLoS ONE, Vol. 1, Núm. 1

2004

  1. Alteration of the bioenergetic phenotype of mitochondria is a hallmark of breast, gastric, lung and oesophageal cancer

    Biochemical Journal, Vol. 378, Núm. 1, pp. 17-20