Publicacións nas que colabora con MARGARITA FERNÁNDEZ GARCÍA DE CASTRO (34)

2018

  1. Bone morphogenetic protein 9 as a key regulator of liver progenitor cells in DDC-induced cholestatic liver injury

    Liver International, Vol. 38, Núm. 9, pp. 1664-1675

2016

  1. Dissecting the role of epidermal growth factor receptor catalytic activity during liver regeneration and hepatocarcinogenesis

    Hepatology, Vol. 63, Núm. 2, pp. 604-619

2015

  1. BMP9-induced survival effect in liver tumor cells requires p38MAPK activation

    International Journal of Molecular Sciences, Vol. 16, Núm. 9, pp. 20431-20448

  2. HGF/c-Met signaling promotes liver progenitor cell migration and invasion by an epithelial-mesenchymal transition-independent, phosphatidyl inositol-3 kinase-dependent pathway in an in vitro model

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1853, Núm. 10, pp. 2453-2463

2014

  1. Polμ deficiency increases resistance to oxidative damage and delays liver aging

    PLoS ONE, Vol. 9, Núm. 4

2013

  1. BMP9 Is a Proliferative and Survival Factor for Human Hepatocellular Carcinoma Cells

    PLoS ONE, Vol. 8, Núm. 7

  2. Glucagon regulation of oxidative phosphorylation requires an increase in matrix adenine nucleotide content through Ca2+ activation of the mitochondrial ATP-Mg/Pi carrier SCaMC-3

    Journal of Biological Chemistry, Vol. 288, Núm. 11, pp. 7791-7802

  3. Mouse Hepatic Oval Cells Require Met-Dependent PI3K to Impair TGF-β-Induced Oxidative Stress and Apoptosis

    PLoS ONE, Vol. 8, Núm. 1

  4. Sterile inflammation in acetaminophen-induced liver injury is mediated by Cot/tpl2

    Journal of Biological Chemistry, Vol. 288, Núm. 21, pp. 15342-15351

2012

  1. EGFR is dispensable for c-Met-mediated proliferation and survival activities in mouse adult liver oval cells

    Cellular Signalling, Vol. 24, Núm. 2, pp. 505-513

2011

  1. Cot/tpl2 activity is required for TLR-induced activation of the Akt p70 S6k pathway in macrophages: Implications for NO synthase 2 expression

    European Journal of Immunology, Vol. 41, Núm. 6, pp. 1733-1741

  2. Protein tyrosine phosphatase 1B (PTP1B) deficiency accelerates hepatic regeneration in mice

    American Journal of Pathology, Vol. 178, Núm. 4, pp. 1591-1604

2008

  1. Deletion of the Met tyrosine kinase in liver progenitor oval cells increases sensitivity to apoptosis in vitro

    American Journal of Pathology, Vol. 172, Núm. 5, pp. 1238-1247

  2. Isolation and characterization of a putative liver progenitor population after treatment of fetal rat hepatocytes with TGF-beta

    Journal of Cellular Physiology, Vol. 215, Núm. 3, pp. 846-855

2007

  1. Activation of NADPH oxidase by transforming growth factor-β in hepatocytes mediates up-regulation of epidermal growth factor receptor ligands through a nuclear factor-κB-dependent mechanism

    Biochemical Journal, Vol. 405, Núm. 2, pp. 251-259

2006

  1. Autocrine production of TGF-β confers resistance to apoptosis after an epithelial-mesenchymal transition process in hepatocytes: Role of EGF receptor ligands

    Experimental Cell Research, Vol. 312, Núm. 15, pp. 2860-2871

  2. EGF blocks NADPH oxidase activation by TGF-β in fetal rat hepatocytes, impairing oxidative stress, and cell death

    Journal of Cellular Physiology, Vol. 207, Núm. 2, pp. 322-330

2004

  1. Resistance to TGF-β-induced apoptosis in regenerating hepatocytes

    Journal of Cellular Physiology, Vol. 201, Núm. 3, pp. 385-392

  2. Source of early reactive oxygen species in the apoptosis induced by transforming growth factor-β in fetal rat hepatocytes

    Free Radical Biology and Medicine, Vol. 36, Núm. 1, pp. 16-26

  3. Transforming growth factor-beta activates both pro-apoptotic and survival signals in fetal rat hepatocytes

    Experimental Cell Research, Vol. 292, Núm. 1, pp. 209-218