Publicacions en què col·labora amb Tomas Sobrino Moreiras (13)

2013

  1. Neuronal excitotoxicity after carotid angioplasty and stent placement procedures

    Radiology, Vol. 268, Núm. 2, pp. 515-520

2011

  1. The natural tissue plasminogen activator inhibitor neuroserpin and acute ischaemic stroke outcome

    Thrombosis and Haemostasis, Vol. 105, Núm. 3, pp. 421-429

2008

  1. Delayed post-ischemic administration of CDP-choline increases EAAT2 association to lipid rafts and affords neuroprotection in experimental stroke

    Neurobiology of Disease, Vol. 29, Núm. 1, pp. 123-131

2007

  1. A chronic treatment with CDP-choline improves functional recovery and increases neuronal plasticity after experimental stroke

    Neurobiology of Disease, Vol. 26, Núm. 1, pp. 105-111

  2. Increased body iron stores are associated with poor outcome after thrombolytic treatment in acute stroke

    Stroke, Vol. 38, Núm. 1, pp. 90-95

  3. Neuroplasticity and cellular therapy in cerebral infarction

    Cerebrovascular Diseases

  4. The increase of circulating endothelial progenitor cells after acute ischemic stroke is associated with good outcome

    Stroke, Vol. 38, Núm. 10, pp. 2759-2764

2006

  1. A polymorphism in the EAAT2 promoter is associated with higher glutamate concentrations and higher frequency of progressing stroke

    Journal of Experimental Medicine, Vol. 203, Núm. 3, pp. 711-717

  2. High blood pressure and inflammation are associated with poor prognosis in lacunar infarctions

    Cerebrovascular Diseases, Vol. 22, Núm. 2-3, pp. 123-129

  3. Hyperthermia is a surrogate marker of inflammationmediated cause of brain damage in acute ischaemic stroke

    Journal of Internal Medicine, Vol. 260, Núm. 4, pp. 343-349

  4. Ischemic preconditioning: A novel target for neuroprotective therapy

    Cerebrovascular Diseases, Vol. 21, Núm. SUPPL. 2, pp. 38-47

  5. Micro- and macroalbuminuria predict hemorrhagic transformation in acute ischemic stroke

    Neurology, Vol. 67, Núm. 7, pp. 1172-1177

  6. Neurorepair versus neuroprotection in stroke

    Cerebrovascular Diseases, Vol. 21, Núm. SUPPL. 2, pp. 54-63