Catalina Picó Segura-rekin lankidetzan egindako argitalpenak (37)

2020

  1. Identification of blood cell transcriptome-based biomarkers in adulthood predictive of increased risk to develop metabolic disorders using early life intervention rat models

    FASEB Journal, Vol. 34, Núm. 7, pp. 9003-9017

2017

  1. Oral leptin supplementation throughout lactation in rats prevents later metabolic alterations caused by gestational calorie restriction

    International Journal of Obesity, Vol. 41, Núm. 3, pp. 360-371

  2. Sexual dimorphism in the age-induced insulin resistance, liver steatosis, and adipose tissue function in rats

    Frontiers in Physiology, Vol. 8, Núm. JUL

2015

  1. Moderate calorie restriction during gestation programs offspring for lower BAT thermogenic capacity driven by thyroid and sympathetic signaling

    International Journal of Obesity, Vol. 39, Núm. 2, pp. 339-345

  2. TAS1R3 and UCN2 transcript levels in blood cells are associated with sugary and fatty food consumption in children

    Journal of Clinical Endocrinology and Metabolism, Vol. 100, Núm. 9, pp. 3556-3564

2014

  1. Influence of breastfeeding on blood-cell transcript-based biomarkers of health in children

    Pediatric Obesity, Vol. 9, Núm. 6, pp. 463-470

  2. Maternal fat supplementation during late pregnancy and lactation influences the development of hepatic steatosis in offspring depending on the fat source

    Journal of Agricultural and Food Chemistry, Vol. 62, Núm. 7, pp. 1590-1601

2013

  1. Early alterations in plasma ghrelin levels in offspring of calorie-restricted rats during gestation may be linked to lower sympathetic drive to the stomach

    Peptides, Vol. 39, Núm. 1, pp. 59-63

  2. Maternal dietary fat affects milk fatty acid profile and impacts on weight gain and thermogenic capacity of suckling rats

    Lipids, Vol. 48, Núm. 5, pp. 481-495

  3. Metabolic programming of sirtuin 1 (SIRT1) expression by moderate energy restriction during gestation in rats may be related to obesity susceptibility in later life

    British Journal of Nutrition, Vol. 109, Núm. 4, pp. 757-764

2012

  1. Blood cells as a source of transcriptional biomarkers of childhood obesity and its related metabolic alterations: Results of the IDEFICS study

    Journal of Clinical Endocrinology and Metabolism, Vol. 97, Núm. 4

  2. Impaired insulin and leptin sensitivity in the offspring of moderate caloric-restricted dams during gestation is early programmed

    Journal of Nutritional Biochemistry, Vol. 23, Núm. 12, pp. 1627-1639

  3. Maternal supplementation with an excess of different fat sources during pregnancy and lactation differentially affects feeding behavior in offspring: Putative role of the leptin system

    Molecular Nutrition and Food Research, Vol. 56, Núm. 11, pp. 1715-1728

  4. Metabolic programming of obesity by energy restriction during the perinatal period: Different outcomes depending on gender and period, type and severity of restriction

    Frontiers in Physiology, Vol. 3 NOV

2011

  1. Moderate caloric restriction during gestation in rats alters adipose tissue sympathetic innervation and later adiposity in offspring

    PLoS ONE, Vol. 6, Núm. 2

  2. Moderate caloric restriction in lactating rats programs their offspring for a better response to HF diet feeding in a sex-dependent manner

    Journal of Nutritional Biochemistry, Vol. 22, Núm. 6, pp. 574-584

  3. Papel de la leptina en la lactancia materna

    Revista Espanola de Nutricion Humana y Dietetica, Vol. 15, Núm. 3, pp. 122-123

  4. Papel de la leptina en la lactancia materna

    Revista española de nutrición humana y dietética, Vol. 15, Núm. 3, pp. 122-123

  5. Protective effects of leptin during the suckling period against later obesity may be associated with changes in promoter methylation of the hypothalamic pro-opiomelanocortin gene

    British Journal of Nutrition, Vol. 106, Núm. 5, pp. 769-778

2010

  1. Leptin intake during the suckling period improves the metabolic response of adipose tissue to a high-fat diet

    International Journal of Obesity, Vol. 34, Núm. 5, pp. 809-819