J. Sáenz Medina-rekin lankidetzan egindako argitalpenak (11)

2023

  1. Hospitalization Burden of Patients with Kidney Stones and Metabolic Comorbidities in Spain during the Period 2017–2020

    Metabolites, Vol. 13, Núm. 4

2022

  1. Differential contribution of renal cytochrome P450 enzymes to kidney endothelial dysfunction and vascular oxidative stress in obesity

    Biochemical Pharmacology, Vol. 195

  2. Endothelial Dysfunction: An Intermediate Clinical Feature between Urolithiasis and Cardiovascular Diseases

    International Journal of Molecular Sciences, Vol. 23, Núm. 2

  3. Hyperoxaluria Induces Endothelial Dysfunction in Preglomerular Arteries: Involvement of Oxidative Stress

    Cells, Vol. 11, Núm. 15

2021

  1. Activation of AMP kinase ameliorates kidney vascular dysfunction, oxidative stress and inflammation in rodent models of obesity

    British Journal of Pharmacology, Vol. 178, Núm. 20, pp. 4085-4103

  2. Urolithiasis develops endothelial dysfunction as a clinical feature

    Antioxidants, Vol. 10, Núm. 5

2020

  1. Activation of the AMP-related kinase (AMPK) induces renal vasodilatation and downregulates Nox-derived reactive oxygen species (ROS) generation

    Redox Biology, Vol. 34

  2. Differential contribution of Nox1, Nox2 and Nox4 to kidney vascular oxidative stress and endothelial dysfunction in obesity

    Redox Biology, Vol. 28

  3. Nox1-derived oxidative stress as a common pathogenic link between obesity and hyperoxaluria-related kidney injury

    Urolithiasis, Vol. 48, Núm. 6, pp. 481-492

2018

  1. Hydrogen peroxide derived from NADPH oxidase 4- and 2 contributes to the endothelium-dependent vasodilatation of intrarenal arteries

    Redox Biology, Vol. 19, pp. 92-104

  2. Metabolic syndrome contributes to renal injury mediated by hyperoxaluria in a murine model of nephrolithiasis

    Urolithiasis, Vol. 46, Núm. 2, pp. 179-186