Publikationen, an denen er mitarbeitet Marta Ruiz Ortega (58)

2022

  1. CCN2 (Cellular Communication Network Factor 2) Deletion Alters Vascular Integrity and Function Predisposing to Aneurysm Formation

    Hypertension, Vol. 79, Núm. 3, pp. E42-E55

  2. Molecular Mechanisms of Kidney Injury and Repair

    International Journal of Molecular Sciences, Vol. 23, Núm. 3

2021

  1. Deletion of delta-like 1 homologue accelerates renal inflammation by modulating the Th17 immune response

    FASEB Journal, Vol. 35, Núm. 1

2020

  1. Interleukin-17A induces vascular remodeling of small arteries and blood pressure elevation

    Clinical Science, Vol. 134, Núm. 5, pp. 513-527

2019

  1. Interleukin 17A participates in renal inflammation associated to experimental and human hypertension

    Frontiers in Pharmacology, Vol. 10

2018

  1. Connective tissue growth factor induces renal fibrosis via epidermal growth factor receptor activation

    Journal of Pathology, Vol. 244, Núm. 2, pp. 227-241

  2. Impact of altered intestinal microbiota on chronic kidney disease progression

    Toxins, Vol. 10, Núm. 7

2017

  1. 2017 update on the relationship between diabetes and colorectal cancer: Epidemiology, potential molecular mechanisms and therapeutic implications

    Oncotarget, Vol. 8, Núm. 11, pp. 18456-18485

  2. Corrigendum to "T Helper 17/Regulatory T Cell Balance and Experimental Models of Peritoneal Dialysis-Induced Damage"

    BioMed research international

2016

  1. Calcineurin inhibitors cyclosporine A and tacrolimus induce vascular inflammation and endothelial activation through TLR4 signaling

    Scientific Reports, Vol. 6

2015

  1. Biocompatibility reduces inflammation-induced apoptosis in mesothelial cells exposed to peritoneal dialysis Fluid

    Blood Purification, Vol. 39, Núm. 1-3, pp. 200-209

  2. Gremlin regulates renal inflammation via the vascular endothelial growth factor receptor 2 pathway

    Journal of Pathology, Vol. 236, Núm. 4, pp. 407-420

  3. T helper 17/regulatory t cell balance and experimental models of peritoneal dialysis-induced damage

    BioMed Research International, Vol. 2015

  4. TGF-Beta Blockade Increases Renal Inflammation Caused by the C-Terminal Module of the CCN2

    Mediators of Inflammation, Vol. 2015

  5. TNF-related weak inducer of apoptosis (TWEAK) regulates junctional proteins in tubular epithelial cells via canonical NF-κB pathway and ERK activation

    Journal of Cellular Physiology, Vol. 230, Núm. 7, pp. 1580-1593

  6. The C-terminal module IV of connective tissue growth factor, through EGFR/Nox1 signaling, activates the NF-κB pathway and proinflammatory factors in vascular smooth muscle cells

    Antioxidants and Redox Signaling, Vol. 22, Núm. 1, pp. 29-47

2014

  1. Endogenous NAMPT dampens chemokine expression and apoptotic responses in stressed tubular cells

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1842, Núm. 2, pp. 293-303

  2. Gremlin activates the smad pathway linked to epithelial mesenchymal transdifferentiation in cultured tubular epithelial cells

    BioMed Research International, Vol. 2014

  3. IL-17A is a novel player in dialysis-induced peritoneal damage

    Kidney International, Vol. 86, Núm. 2, pp. 303-315

  4. Integrin-linked kinase plays a key role in the regulation of angiotensin II-induced renal inflammation

    Clinical Science, Vol. 127, Núm. 1, pp. 19-31