Jesús Egido de los Ríos-rekin lankidetzan egindako argitalpenak (44)

1997

  1. The 80-kD fibronectin fragment increases the production of fibronectin and tumour necrosis factor-alpha (TNF-α) in cultured mesangial cells

    Clinical and Experimental Immunology, Vol. 107, Núm. 2, pp. 398-403

1996

  1. Anti-inflammatory effect of a PAF receptor antagonist and a new molecule with antiproteinase activity in an experimental model of acute urate crystal arthritis

    Journal of Lipid Mediators and Cell Signalling, Vol. 13, Núm. 1, pp. 35-49

  2. Effects and interactions of endothelin-1 and angiotensin II on matrix protein expression and synthesis and mesangial cell growth

    Hypertension, Vol. 27, Núm. 4, pp. 885-892

  3. Endothelin-1 induces loss of proteoglycans and enhances fibronectin and collagen production in cultured rabbit synovial cells

    European Journal of Pharmacology, Vol. 302, Núm. 1-3, pp. 191-197

  4. Interferon-inducible protein-10 is highly expressed in rats with experimental nephrosis

    American Journal of Pathology, Vol. 148, Núm. 1, pp. 301-311

  5. Urinary excretion of platelet-activating factor in human and experimental nephrosis

    Nephrology Dialysis Transplantation, Vol. 11, Núm. 2, pp. 282-286

1995

  1. ACE inhibition reduces proteinuria, glomerular lesions and extracellular matrix production in a normotensive rat model of immune complex nephritis

    Kidney International, Vol. 48, Núm. 6, pp. 1778-1791

  2. Cyclosporin A (CsA) modulates the glomerular production of inflammatory mediators and proteoglycans in experimental nephrosis

    Clinical and Experimental Immunology, Vol. 102, Núm. 3, pp. 608-613

  3. Fibronectin (FN) decreases glomerular lesions and synthesis of tumour necrosis factor-alpha (TNF-α), platelet-activating factor (PAF) and FN in proliferative glomerulonephritis

    Clinical and Experimental Immunology, Vol. 101, Núm. 2, pp. 334-340

  4. IL-1β and IL-6 stimulate the production of platelet-activating factor (PAF) by cultured rabbit synovial cells

    Clinical and Experimental Immunology, Vol. 99, Núm. 3, pp. 364-368

  5. Involvement of tumor necrosis factor-alpha in the pathogenesis of experimental and human glomerulonephritis.

    Advances in nephrology from the Necker Hospital, Vol. 24, pp. 53-77

  6. Potential factors governing extracellular matrix production by mesangial cells: their relevance for the pathogenesis of IgA nephropathy.

    Contributions to nephrology, Vol. 111, pp. 45-54

1994

  1. Increase of tumour necrosis factor α synthesis and gene expression in peripheral blood mononuclear cells of children with idiopathic nephrotic syndrome

    European Journal of Clinical Investigation, Vol. 24, Núm. 12, pp. 799-805

  2. Involvement of angiotensin ii and endothelin in matrix protein production and renal sclerosis

    Journal of Hypertension, Vol. 12, pp. S51-S58

  3. Involvement of tumor necrosis factor and platelet-activating factor in the pathogenesis of experimental nephrosis in rats

    Laboratory Investigation, Vol. 70, Núm. 4, pp. 449-459

  4. Long-term effect of nonsteroidal anti-inflammatory drugs on the production of cytokines and other inflammatory mediators by blood cells of patients with osteoarthritis

    Agents and Actions, Vol. 41, Núm. 3-4, pp. 171-178

  5. Soluble IgA and IgG aggregates are catabolized by cultured rat mesangial cells and induce production of TNF-α and IL-6, and proliferation

    Journal of Immunology, Vol. 153, Núm. 11, pp. 5247-5256

  6. The potential role of inflammatory and fibrogenic cytokines in the glomerular diseases

    Journal of Lipid Mediators and Cell Signalling, Vol. 9, Núm. 1, pp. 55-74

1993

  1. Evidence for a specific IgA receptor in rat and human mesangial cells

    Journal of Immunology, Vol. 151, Núm. 12, pp. 7172-7181

  2. Interaction of mesangial cells with IgA and IgG immune complexes: a possible mechanism of glomerular injury in IgA nephropathy.

    Contributions to nephrology, Vol. 104, pp. 127-137