Publicaciones en colaboración con investigadores/as de Imperial College London (25)

2024

  1. Prevalence of comorbidities with the potential to increase the risk of nonadherence to topical ocular hypotensive medication in patients with open-angle glaucoma

    Current Medical Research and Opinion

2023

  1. A second update on mapping the human genetic architecture of COVID-19

    Nature

  2. Correction: “The 5th edition of The World Health Organization Classification of Haematolymphoid Tumours: Lymphoid Neoplasms” Leukemia. 2022 Jul;36(7):1720–1748 (Leukemia, (2022), 36, 7, (1720-1748), 10.1038/s41375-022-01620-2)

    Leukemia

  3. Genetic signature detected in T cell receptors from patients with severe COVID-19

    iScience, Vol. 26, Núm. 10

2022

  1. Factor H-Related Protein 1 Drives Disease Susceptibility and Prognosis in C3 Glomerulopathy

    Journal of the American Society of Nephrology : JASN, Vol. 33, Núm. 6, pp. 1137-1153

  2. Human genetic and immunological determinants of critical COVID-19 pneumonia

    Nature, Vol. 603, Núm. 7902, pp. 587-598

  3. New molecular mechanisms in cholangiocarcinoma: signals triggering interleukin-6 production in tumor cells and KRAS co-opted epigenetic mediators driving metabolic reprogramming

    Journal of Experimental and Clinical Cancer Research, Vol. 41, Núm. 1

2020

  1. Autoantibodies against type I IFNs in patients with life-threatening COVID-19

    Science, Vol. 370, Núm. 6515

  2. Inborn errors of type I IFN immunity in patients with life-threatening COVID-19

    Science, Vol. 370, Núm. 6515

2019

  1. Editorial: Reverse Vaccinology

    Frontiers in Immunology

  2. The role of complement in IgA nephropathy

    Molecular Immunology, Vol. 114, pp. 123-132

2018

  1. Priming of dendritic cells by DNA-containing extracellular vesicles from activated T cells through antigen-driven contacts

    Nature Communications, Vol. 9, Núm. 1

2015

  1. Factor H-related protein 5 interacts with pentraxin 3 and the extracellular matrix and modulates complement activation

    Journal of Immunology, Vol. 194, Núm. 10, pp. 4963-4973

  2. Factor H-related proteins determine complement-activating surfaces

    Trends in Immunology, Vol. 36, Núm. 6, pp. 374-384

2013

  1. C3 glomerulopathy-associated CFHR1 mutation alters FHR oligomerization and complement regulation

    Journal of Clinical Investigation, Vol. 123, Núm. 6, pp. 2434-2446

  2. C3 glomerulopathy: Consensus report

    Kidney International

  3. Dimerization of complement factor H-related proteins modulates complement activation in vivo

    Proceedings of the National Academy of Sciences of the United States of America, Vol. 110, Núm. 12, pp. 4685-4690

2012

  1. A hybrid CFHR3-1 gene causes familial C3 glomerulopathy

    Journal of the American Society of Nephrology, Vol. 23, Núm. 7, pp. 1155-1160

  2. Acute presentation and persistent glomerulonephritis following streptococcal infection in a patient with heterozygous complement factor H-related protein 5 deficiency

    American Journal of Kidney Diseases, Vol. 60, Núm. 1, pp. 121-125

  3. Design and Evaluation of Meningococcal Vaccines through Structure-Based Modification of Host and Pathogen Molecules

    PLoS Pathogens, Vol. 8, Núm. 10