Medicina Legal, Psiquiatría y Patología
Departamento
Pilar
Sánchez Gómez
Publicaciones en las que colabora con Pilar Sánchez Gómez (14)
2022
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Blood-Brain Barrier Disruption: A Common Driver of Central Nervous System Diseases
Neuroscientist, Vol. 28, Núm. 3, pp. 222-237
2021
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Tumor-derived pericytes driven by egfr mutations govern the vascular and immune microenvironment of gliomas
Cancer Research, Vol. 81, Núm. 8, pp. 2142-2156
2020
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Immune profiling of gliomas reveals a connection with IDH1/2 mutations, tau function and the vascular phenotype
Cancers, Vol. 12, Núm. 11, pp. 1-18
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Phase II Trial of Palbociclib in Recurrent Retinoblastoma-Positive Anaplastic Oligodendroglioma: A Study from the Spanish Group for Research in Neuro-Oncology (GEINO)
Targeted Oncology, Vol. 15, Núm. 5, pp. 613-622
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The EGFR-TMEM167A-p53 axis defines the aggressiveness of gliomas
Cancers, Vol. 12, Núm. 1
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The IDH-TAU-EGFR triad defines the neovascular landscape of diffuse gliomas
Science Translational Medicine, Vol. 12, Núm. 527
2019
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Correlation of radiological and immunochemical parameters with clinical outcome in patients with recurrent glioblastoma treated with Bevacizumab
Clinical and Translational Oncology, Vol. 21, Núm. 10, pp. 1413-1423
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NFATc3 controls tumour growth by regulating proliferation and migration of human astroglioma cells
Scientific Reports, Vol. 9, Núm. 1
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Ocoxin Modulates Cancer Stem Cells and M2 Macrophage Polarization in Glioblastoma
Oxidative medicine and cellular longevity, Vol. 2019, pp. 9719730
2017
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Glioblastoma on a microfluidic chip: Generating pseudopalisades and enhancing aggressiveness through blood vessel obstruction events
Neuro-Oncology, Vol. 19, Núm. 4, pp. 503-513
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Phase II trial of dacomitinib, a pan-human EGFR tyrosine kinase inhibitor, in recurrent glioblastoma patients with EGFR amplification
Neuro-Oncology, Vol. 19, Núm. 11, pp. 1522-1531
2015
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Controlled release microspheres loaded with BMP7 suppress primary tumors from human glioblastoma
Oncotarget, Vol. 6, Núm. 13, pp. 10950-10963
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Preclinical test of dacomitinib, an irreversible EGFR inhibitor, confirms its effectiveness for glioblastoma
Molecular Cancer Therapeutics, Vol. 14, Núm. 7, pp. 1548-1558
2013
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Inhibition of DYRK1A destabilizes EGFR and reduces EGFR-dependent glioblastoma growth
Journal of Clinical Investigation, Vol. 123, Núm. 6, pp. 2475-2487