Medicina Legal, Psiquiatría y Patología
Departamento
PAULA VIVIANA
MOYANO-CIRES IVANOFF
Profesora ayudante doctora
Publicaciones en las que colabora con PAULA VIVIANA MOYANO-CIRES IVANOFF (31)
2024
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Imidacloprid unique and repeated treatment produces cholinergic transmission disruption and apoptotic cell death in SN56 cells
Food and Chemical Toxicology, Vol. 193
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Increased Levels of Phosphorylated-P38α Induce WNT/β-Catenin and NGF/P75NTR/TrkA Pathways Disruption and SN56 Cell Death following Single and Repeated Chlorpyrifos Treatment
Foods, Vol. 13, Núm. 15
2023
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Bisphenol-A Neurotoxic Effects on Basal Forebrain Cholinergic Neurons In Vitro and In Vivo
Biology, Vol. 12, Núm. 6
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Cadmium-promoted thyroid hormones disruption mediates ROS, inflammation, Aβ and Tau proteins production, gliosis, spongiosis and neurodegeneration in rat basal forebrain
Chemico-Biological Interactions, Vol. 375
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Neurodegenerative Proteinopathies Induced by Environmental Pollutants: Heat Shock Proteins and Proteasome as Promising Therapeutic Tools
Pharmaceutics, Vol. 15, Núm. 8
2022
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Cadmium-induced neurotoxic effects on rat basal forebrain cholinergic system through thyroid hormones disruption
Environmental Toxicology and Pharmacology, Vol. 90
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Insulin Signaling Disruption and INF-γUpregulation Induce Aβ1-42and Hyperphosphorylated-Tau Proteins Synthesis and Cell Death after Paraquat Treatment of Primary Hippocampal Cells
Chemical Research in Toxicology, Vol. 35, Núm. 12, pp. 2214-2218
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Single and repeated bisphenol A treatment induces ROS, Aβ and hyperphosphorylated-tau accumulation, and insulin pathways disruption, through HDAC2 and PTP1B overexpression, leading to SN56 cholinergic apoptotic cell death
Food and Chemical Toxicology, Vol. 170
2021
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Bisphenol A single and repeated treatment increases HDAC2, leading to cholinergic neurotransmission dysfunction and SN56 cholinergic apoptotic cell death through AChE variants overexpression and NGF/TrkA/P75NTR signaling disruption
Food and Chemical Toxicology, Vol. 157
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Chlorpyrifos induces cell proliferation in MCF-7 and MDA-MB-231 cells, through cholinergic and Wnt/β-catenin signaling disruption, AChE-R upregulation and oxidative stress generation after single and repeated treatment
Food and Chemical Toxicology, Vol. 152
2020
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Chlorpyrifos-induced cell proliferation in human breast cancer cell lines differentially mediated by estrogen and aryl hydrocarbon receptors and KIAA1363 enzyme after 24 h and 14 days exposure
Chemosphere, Vol. 251
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Dysregulation of prostaglandine E2 and BDNF signaling mediated by estrogenic dysfunction induces primary hippocampal neuronal cell death after single and repeated paraquat treatment
Food and Chemical Toxicology, Vol. 144
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Manganese increases Aβ and Tau protein levels through proteasome 20S and heat shock proteins 90 and 70 alteration, leading to SN56 cholinergic cell death following single and repeated treatment
Ecotoxicology and Environmental Safety, Vol. 203
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Primary hippocampal estrogenic dysfunction induces synaptic proteins alteration and neuronal cell death after single and repeated paraquat exposure
Food and Chemical Toxicology, Vol. 136
2019
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Manganese induced ROS and AChE variants alteration leads to SN56 basal forebrain cholinergic neuronal loss after acute and long-term treatment
Food and Chemical Toxicology, Vol. 125, pp. 583-594
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Oxidative stress and cell death induction by amitraz and its metabolite BTS-27271 mediated through cytochrome P450 and NRF2 pathway alteration in primary hippocampal cell
Food and Chemical Toxicology, Vol. 129, pp. 87-96
2018
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Cadmium alters heat shock protein pathways in SN56 cholinergic neurons, leading to Aβ and phosphorylated Tau protein generation and cell death
Food and Chemical Toxicology, Vol. 121, pp. 297-308
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Cadmium induced ROS alters M1 and M3 receptors, leading to SN56 cholinergic neuronal loss, through AChE variants disruption
Toxicology, Vol. 394, pp. 54-62
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SN56 neuronal cell death after 24 h and 14 days chlorpyrifos exposure through glutamate transmission dysfunction, increase of GSK-3β enzyme, β-amyloid and tau protein levels
Toxicology, Vol. 402-403, pp. 17-27
2017
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Amitraz changes NE, DA and 5-HT biosynthesis and metabolism mediated by alterations in estradiol content in CNS of male rats
Chemosphere, Vol. 181, pp. 518-529