Instituto Universitario de Investigación en Neuroquímica
Centro/Instituto
![Foto de José Antonio](/img/nophoto.png)
José Antonio
Castillo Sánchez
Publicaciones en las que colabora con José Antonio Castillo Sánchez (29)
2013
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Neuronal excitotoxicity after carotid angioplasty and stent placement procedures
Radiology, Vol. 268, Núm. 2, pp. 515-520
2011
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The natural tissue plasminogen activator inhibitor neuroserpin and acute ischaemic stroke outcome
Thrombosis and Haemostasis, Vol. 105, Núm. 3, pp. 421-429
2008
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Delayed post-ischemic administration of CDP-choline increases EAAT2 association to lipid rafts and affords neuroprotection in experimental stroke
Neurobiology of Disease, Vol. 29, Núm. 1, pp. 123-131
2007
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A chronic treatment with CDP-choline improves functional recovery and increases neuronal plasticity after experimental stroke
Neurobiology of Disease, Vol. 26, Núm. 1, pp. 105-111
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Increased body iron stores are associated with poor outcome after thrombolytic treatment in acute stroke
Stroke, Vol. 38, Núm. 1, pp. 90-95
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Neuroplasticity and cellular therapy in cerebral infarction
Cerebrovascular Diseases
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Statin treatment withdrawal in ischemic stroke: A controlled randomized study
Neurology, Vol. 69, Núm. 9, pp. 904-910
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The increase of circulating endothelial progenitor cells after acute ischemic stroke is associated with good outcome
Stroke, Vol. 38, Núm. 10, pp. 2759-2764
2006
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A polymorphism in the EAAT2 promoter is associated with higher glutamate concentrations and higher frequency of progressing stroke
Journal of Experimental Medicine, Vol. 203, Núm. 3, pp. 711-717
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High blood pressure and inflammation are associated with poor prognosis in lacunar infarctions
Cerebrovascular Diseases, Vol. 22, Núm. 2-3, pp. 123-129
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Hyperthermia is a surrogate marker of inflammationmediated cause of brain damage in acute ischaemic stroke
Journal of Internal Medicine, Vol. 260, Núm. 4, pp. 343-349
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Ischemic preconditioning: A novel target for neuroprotective therapy
Cerebrovascular Diseases, Vol. 21, Núm. SUPPL. 2, pp. 38-47
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Micro- and macroalbuminuria predict hemorrhagic transformation in acute ischemic stroke
Neurology, Vol. 67, Núm. 7, pp. 1172-1177
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Neurorepair versus neuroprotection in stroke
Cerebrovascular Diseases, Vol. 21, Núm. SUPPL. 2, pp. 54-63
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New-onset hypertension and inflammatory response/poor outcome in acute ischemic stroke
Neurology, Vol. 67, Núm. 11, pp. 1973-1978
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Rosiglitazone and 15-deoxy-Δ12,14-prostaglandin J 2 cause potent neuroprotection after experimental stroke through noncompletely overlapping mechanisms
Journal of Cerebral Blood Flow and Metabolism, Vol. 26, Núm. 2, pp. 218-229
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Targets of cytoprotection in acute ischemic stroke: Present and future
Cerebrovascular Diseases, Vol. 21, Núm. SUPPL. 2, pp. 1-8
2005
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Increased plasma levels of 15-deoxy Δ prostaglandin J2 are associated with good outcome in acute atherothrombotic ischemic stroke
Stroke, Vol. 36, Núm. 6, pp. 1189-1194
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Inhibition of iNOS activity by 1400W decreases glutamate release and ameliorates stroke outcome after experimental ischemia
Neurobiology of Disease, Vol. 18, Núm. 2, pp. 375-384
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Neuroprotection afforded by prior citicoline administration in experimental brain ischemia: Effects on glutamate transport
Neurobiology of Disease, Vol. 18, Núm. 2, pp. 336-345