Publikationen, an denen er mitarbeitet MARÍA ISABEL FABREGAT ROMERO (72)

2022

  1. Lack of EGFR catalytic activity in hepatocytes improves liver regeneration following DDC-induced cholestatic injury by promoting a pro-restorative inflammatory response

    Journal of Pathology, Vol. 258, Núm. 3, pp. 312-324

  2. Mitochondrial bioenergetics boost macrophage activation, promoting liver regeneration in metabolically compromised animals

    Hepatology, Vol. 75, Núm. 3, pp. 550-566

2020

  1. A Signaling Crosstalk between BMP9 and HGF/c-Met Regulates Mouse Adult Liver Progenitor Cell Survival

    Cells, Vol. 9, Núm. 3

  2. Case Report: An EGFR-Targeted 4-1BB-agonistic Trimerbody Does Not Induce Hepatotoxicity in Transgenic Mice With Liver Expression of Human EGFR

    Frontiers in Immunology, Vol. 11

  3. Clathrin switches transforming growth factor-β role to pro-tumorigenic in liver cancer

    Journal of Hepatology, Vol. 72, Núm. 1, pp. 125-134

  4. Editorial Special Issue TGF-beta/BMP Signaling Pathway

    Cells

2019

  1. c-Met Signaling Is Essential for Mouse Adult Liver Progenitor Cells Expansion After Transforming Growth Factor-β-Induced Epithelial–Mesenchymal Transition and Regulates Cell Phenotypic Switch

    Stem Cells, Vol. 37, Núm. 8, pp. 1108-1118

2018

  1. Bone morphogenetic protein 9 as a key regulator of liver progenitor cells in DDC-induced cholestatic liver injury

    Liver International, Vol. 38, Núm. 9, pp. 1664-1675

  2. Corrigendum to “European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS)” (Redox Biol. (2017) 13 (94–162)(S2213231717303373)(10.1016/j.redox.2017.05.007))

    Redox Biology

2017

  1. European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS)

    Redox Biology, Vol. 13, pp. 94-162

2016

  1. Caveolin-1-dependent activation of the metalloprotease TACE/ADAM17 by TGF-β in hepatocytes requires activation of Src and the NADPH oxidase NOX1

    The FEBS journal, Vol. 283, Núm. 7, pp. 1300-1310

  2. Dissecting the role of epidermal growth factor receptor catalytic activity during liver regeneration and hepatocarcinogenesis

    Hepatology, Vol. 63, Núm. 2, pp. 604-619

  3. TGF-β signalling and liver disease

    FEBS Journal, pp. 2219-2232

  4. The rationale for targeting TGF-β in chronic liver diseases

    European Journal of Clinical Investigation, Vol. 46, Núm. 4, pp. 349-361

2015

  1. BMP9-induced survival effect in liver tumor cells requires p38MAPK activation

    International Journal of Molecular Sciences, Vol. 16, Núm. 9, pp. 20431-20448

  2. HGF/c-Met signaling promotes liver progenitor cell migration and invasion by an epithelial-mesenchymal transition-independent, phosphatidyl inositol-3 kinase-dependent pathway in an in vitro model

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1853, Núm. 10, pp. 2453-2463

2014

  1. Caveolin-1 is required for TGF-ß-induced transactivation of the EGF receptor pathway in hepatocytes through the activation of the metalloprotease TACE/ADAM17

    Cell Death and Disease, Vol. 5, Núm. 7

  2. Polμ deficiency increases resistance to oxidative damage and delays liver aging

    PLoS ONE, Vol. 9, Núm. 4

  3. The NADPH oxidase NOX4 inhibits hepatocyte proliferation and liver cancer progression

    Free Radical Biology and Medicine, Vol. 69, pp. 338-347

2013

  1. BMP9 Is a Proliferative and Survival Factor for Human Hepatocellular Carcinoma Cells

    PLoS ONE, Vol. 8, Núm. 7