Publicaciones en colaboración con investigadores/as de Helmholtz Zentrum München (14)

2022

  1. Drivers of the release of the allergens Bet v 1 and Phl p 5 from birch and grass pollen

    Environmental Research, Vol. 214

2021

  1. Consequences of climate change on airborne pollen in Bavaria, Central Europe

    Regional Environmental Change, Vol. 21, Núm. 1

  2. Effects of future climate change on birch abundance and their pollen load

    Global Change Biology, Vol. 27, Núm. 22, pp. 5934-5949

  3. Heterogeneous contributions of change in population distribution of body mass index to change in obesity and underweight

    eLife, Vol. 10

  4. Small extracellular vesicle-mediated targeting of hypothalamic AMPKα1 corrects obesity through BAT activation

    Nature Metabolism, Vol. 3, Núm. 10, pp. 1415-1431

2020

  1. HYPOTHesizing about central comBAT against obesity

    Journal of Physiology and Biochemistry, Vol. 76, Núm. 2, pp. 193-211

  2. Land-use and height of pollen sampling affect pollen exposure in Munich, Germany

    Atmosphere, Vol. 11, Núm. 2

2019

  1. Deep Learning Reveals Cancer Metastasis and Therapeutic Antibody Targeting in the Entire Body

    Cell, Vol. 179, Núm. 7, pp. 1661-1676.e19

  2. Glucagon, GLP-1 and thermogenesis

    International Journal of Molecular Sciences, Vol. 20, Núm. 14

  3. Near-ground effect of height on pollen exposure

    Environmental Research, Vol. 174, pp. 160-169

2018

  1. Corrigendum to “European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS)” (Redox Biol. (2017) 13 (94–162)(S2213231717303373)(10.1016/j.redox.2017.05.007))

    Redox Biology

  2. Genome-wide Analyses Identify KIF5A as a Novel ALS Gene

    Neuron, Vol. 97, Núm. 6, pp. 1268-1283.e6

2017

  1. European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS)

    Redox Biology, Vol. 13, pp. 94-162

2016

  1. NEK1 variants confer susceptibility to amyotrophic lateral sclerosis

    Nature Genetics, Vol. 48, Núm. 9, pp. 1037-1042