Mecanismos de señalización celular en enfermedades hepáticas, cardiovasculares y cáncer
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Institut d'Investigació Biomédica de Bellvitge
Barcelona, EspañaPublicaciones en colaboración con investigadores/as de Institut d'Investigació Biomédica de Bellvitge (75)
2024
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The hepatocyte epidermal growth factor receptor (EGFR) pathway regulates the cellular interactome within the liver fibrotic niche
Journal of Pathology, Vol. 263, Núm. 4-5, pp. 482-495
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Uncovering a Novel Functional Interaction Between Adult Hepatic Progenitor Cells, Inflammation and EGFR Signaling During Bile Acids-Induced Injury
International Journal of Biological Sciences, Vol. 20, Núm. 7, pp. 2339-2355
2023
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Autotaxin inhibitor IOA-289 reduces gastrointestinal cancer progression in preclinical models
Journal of Experimental and Clinical Cancer Research, Vol. 42, Núm. 1
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Correction: Autotaxin inhibitor IOA-289 reduces gastrointestinal cancer progression in preclinical models (Journal of Experimental & Clinical Cancer Research, (2023), 42, 1, (197), 10.1186/s13046-023-02780-4)
Journal of Experimental and Clinical Cancer Research
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Dissecting the role of the NADPH oxidase NOX4 in TGF-beta signaling in hepatocellular carcinoma
Redox Biology, Vol. 65
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The NADPH oxidase NOX4 regulates redox and metabolic homeostasis preventing HCC progression
Hepatology, Vol. 78, Núm. 2, pp. 416-433
2022
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Lack of EGFR catalytic activity in hepatocytes improves liver regeneration following DDC-induced cholestatic injury by promoting a pro-restorative inflammatory response
Journal of Pathology, Vol. 258, Núm. 3, pp. 312-324
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Mitochondrial bioenergetics boost macrophage activation, promoting liver regeneration in metabolically compromised animals
Hepatology, Vol. 75, Núm. 3, pp. 550-566
2021
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2-[18f]fdg pet/ct as a predictor of microvascular invasion and a high histological grade in patients with a hepatocellular carcinoma
Cancers, Vol. 13, Núm. 11
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Anti-miR-518d-5p overcomes liver tumor cell death resistance through mitochondrial activity
Cell Death and Disease, Vol. 12, Núm. 6
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Epithelial–mesenchymal transition (Emt) induced by tgf-β in hepatocellular carcinoma cells reprograms lipid metabolism
International Journal of Molecular Sciences, Vol. 22, Núm. 11
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The tgf-β /nadph oxidases axis in the regulation of liver cell biology in health and disease
Cells, Vol. 10, Núm. 9
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The tgf-β pathway: A pharmacological target in hepatocellular carcinoma?
Cancers, Vol. 13, Núm. 13
2020
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Clathrin switches transforming growth factor-β role to pro-tumorigenic in liver cancer
Journal of Hepatology, Vol. 72, Núm. 1, pp. 125-134
2019
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Calcium regulates HCC proliferation as well as EGFR recycling/degradation and could be a new therapeutic target in HCC
Cancers, Vol. 11, Núm. 10
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Paradoxical role of the NADPH oxidase NOX4 in early preneoplastic stages of hepatocytes induced by amino acid deprivation
Biochimica et Biophysica Acta - General Subjects, Vol. 1863, Núm. 4, pp. 714-722
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c-Met Signaling Is Essential for Mouse Adult Liver Progenitor Cells Expansion After Transforming Growth Factor-β-Induced Epithelial–Mesenchymal Transition and Regulates Cell Phenotypic Switch
Stem Cells, Vol. 37, Núm. 8, pp. 1108-1118
2018
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Altered TGF-β endocytic trafficking contributes to the increased signaling in Marfan syndrome
Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1864, Núm. 2, pp. 554-562
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Bone morphogenetic protein 9 as a key regulator of liver progenitor cells in DDC-induced cholestatic liver injury
Liver International, Vol. 38, Núm. 9, pp. 1664-1675
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Corrigendum to “European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS)” (Redox Biol. (2017) 13 (94–162)(S2213231717303373)(10.1016/j.redox.2017.05.007))
Redox Biology