DOLORES
PRIETO OCEJO
Catedrática de universidad
Publicaciones (147) Publicaciones de DOLORES PRIETO OCEJO
2024
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Activation of mitoKATP channels induces penile vasodilation and inhibits mitochondrial respiration and ROS production: Role of NO
Free Radical Biology and Medicine, Vol. 217, pp. 15-28
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Cardiovascular and Cerebrovascular Morbidity in Patients with Urolithiasis: An Epidemiological Approach Based on Hospitalization Burden Data from 1997 to 2021
Journal of Clinical Medicine, Vol. 13, Núm. 12
2023
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Calcium handling coupled to the endothelin ETA and ETB receptor-mediated vasoconstriction in resistance arteries: Differential regulation by PI3K, PKC and RhoK
European Journal of Pharmacology, Vol. 956
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Hospitalization Burden of Patients with Kidney Stones and Metabolic Comorbidities in Spain during the Period 2017–2020
Metabolites, Vol. 13, Núm. 4
2022
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Differential contribution of renal cytochrome P450 enzymes to kidney endothelial dysfunction and vascular oxidative stress in obesity
Biochemical Pharmacology, Vol. 195
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Endothelial Dysfunction: An Intermediate Clinical Feature between Urolithiasis and Cardiovascular Diseases
International Journal of Molecular Sciences, Vol. 23, Núm. 2
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Hyperoxaluria Induces Endothelial Dysfunction in Preglomerular Arteries: Involvement of Oxidative Stress
Cells, Vol. 11, Núm. 15
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In vitro inhibition of phosphodiesterase type 4 enhances rat corpus cavernosum nerve-mediated relaxation induced by gasotransmitters
Life Sciences, Vol. 296
2021
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AMPK, metabolism, and vascular function
FEBS Journal, Vol. 288, Núm. 12, pp. 3746-3771
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Activation of AMP kinase ameliorates kidney vascular dysfunction, oxidative stress and inflammation in rodent models of obesity
British Journal of Pharmacology, Vol. 178, Núm. 20, pp. 4085-4103
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Endothelial KCa1.1 and KCa3.1 channels mediate rat intrarenal artery endothelium-derived hyperpolarization response
Acta Physiologica, Vol. 231, Núm. 4
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Urolithiasis develops endothelial dysfunction as a clinical feature
Antioxidants, Vol. 10, Núm. 5
2020
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Activation of the AMP-related kinase (AMPK) induces renal vasodilatation and downregulates Nox-derived reactive oxygen species (ROS) generation
Redox Biology, Vol. 34
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Differential contribution of Nox1, Nox2 and Nox4 to kidney vascular oxidative stress and endothelial dysfunction in obesity
Redox Biology, Vol. 28
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Metabolic syndrome inhibits store-operated Ca2+ entry and calcium-induced calcium-release mechanism in coronary artery smooth muscle
Biochemical Pharmacology, Vol. 182
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Nox1-derived oxidative stress as a common pathogenic link between obesity and hyperoxaluria-related kidney injury
Urolithiasis, Vol. 48, Núm. 6, pp. 481-492
2019
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Bladder Dysfunction in an Obese Zucker Rat: The Role of TRPA1 Channels, Oxidative Stress, and Hydrogen Sulfide
Oxidative medicine and cellular longevity, Vol. 2019, pp. 5641645
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Especies reactivas de oxígeno: papel en la función vascular y en la disfunción endotelial asociada a la enfermedad metabólica
Anales de la Real Academia Nacional de Farmacia, Vol. 85, Núm. 4, pp. 288-300
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Role of phosphatidylinositol 3-kinase (PI3K), mitogen-activated protein kinase (MAPK), and protein kinase C (PKC) in calcium signaling pathways linked to the α1-adrenoceptor in resistance arteries
Frontiers in Physiology, Vol. 10, Núm. FEB
2018
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Fisiología de la lactación
Fisiología veterinaria (Tébar Flores), pp. 1009-1030