Publikationen (147) Publikationen von DOLORES PRIETO OCEJO

2024

  1. Activation of mitoKATP channels induces penile vasodilation and inhibits mitochondrial respiration and ROS production: Role of NO

    Free Radical Biology and Medicine, Vol. 217, pp. 15-28

  2. Cardiovascular and Cerebrovascular Morbidity in Patients with Urolithiasis: An Epidemiological Approach Based on Hospitalization Burden Data from 1997 to 2021

    Journal of Clinical Medicine, Vol. 13, Núm. 12

2023

  1. Calcium handling coupled to the endothelin ETA and ETB receptor-mediated vasoconstriction in resistance arteries: Differential regulation by PI3K, PKC and RhoK

    European Journal of Pharmacology, Vol. 956

  2. Hospitalization Burden of Patients with Kidney Stones and Metabolic Comorbidities in Spain during the Period 2017–2020

    Metabolites, Vol. 13, Núm. 4

2022

  1. Differential contribution of renal cytochrome P450 enzymes to kidney endothelial dysfunction and vascular oxidative stress in obesity

    Biochemical Pharmacology, Vol. 195

  2. Endothelial Dysfunction: An Intermediate Clinical Feature between Urolithiasis and Cardiovascular Diseases

    International Journal of Molecular Sciences, Vol. 23, Núm. 2

  3. Hyperoxaluria Induces Endothelial Dysfunction in Preglomerular Arteries: Involvement of Oxidative Stress

    Cells, Vol. 11, Núm. 15

  4. In vitro inhibition of phosphodiesterase type 4 enhances rat corpus cavernosum nerve-mediated relaxation induced by gasotransmitters

    Life Sciences, Vol. 296

2021

  1. AMPK, metabolism, and vascular function

    FEBS Journal, Vol. 288, Núm. 12, pp. 3746-3771

  2. Activation of AMP kinase ameliorates kidney vascular dysfunction, oxidative stress and inflammation in rodent models of obesity

    British Journal of Pharmacology, Vol. 178, Núm. 20, pp. 4085-4103

  3. Endothelial KCa1.1 and KCa3.1 channels mediate rat intrarenal artery endothelium-derived hyperpolarization response

    Acta Physiologica, Vol. 231, Núm. 4

  4. Urolithiasis develops endothelial dysfunction as a clinical feature

    Antioxidants, Vol. 10, Núm. 5

2020

  1. Activation of the AMP-related kinase (AMPK) induces renal vasodilatation and downregulates Nox-derived reactive oxygen species (ROS) generation

    Redox Biology, Vol. 34

  2. Differential contribution of Nox1, Nox2 and Nox4 to kidney vascular oxidative stress and endothelial dysfunction in obesity

    Redox Biology, Vol. 28

  3. Metabolic syndrome inhibits store-operated Ca2+ entry and calcium-induced calcium-release mechanism in coronary artery smooth muscle

    Biochemical Pharmacology, Vol. 182

  4. Nox1-derived oxidative stress as a common pathogenic link between obesity and hyperoxaluria-related kidney injury

    Urolithiasis, Vol. 48, Núm. 6, pp. 481-492

2019

  1. Bladder Dysfunction in an Obese Zucker Rat: The Role of TRPA1 Channels, Oxidative Stress, and Hydrogen Sulfide

    Oxidative medicine and cellular longevity, Vol. 2019, pp. 5641645

  2. Especies reactivas de oxígeno: papel en la función vascular y en la disfunción endotelial asociada a la enfermedad metabólica

    Anales de la Real Academia Nacional de Farmacia, Vol. 85, Núm. 4, pp. 288-300

  3. Role of phosphatidylinositol 3-kinase (PI3K), mitogen-activated protein kinase (MAPK), and protein kinase C (PKC) in calcium signaling pathways linked to the α1-adrenoceptor in resistance arteries

    Frontiers in Physiology, Vol. 10, Núm. FEB

2018

  1. Fisiología de la lactación

    Fisiología veterinaria (Tébar Flores), pp. 1009-1030