JAVIER DEL PINO SANS-rekin lankidetzan egindako argitalpenak (16)

2023

  1. Bisphenol-A Neurotoxic Effects on Basal Forebrain Cholinergic Neurons In Vitro and In Vivo

    Biology, Vol. 12, Núm. 6

  2. Cadmium-promoted thyroid hormones disruption mediates ROS, inflammation, Aβ and Tau proteins production, gliosis, spongiosis and neurodegeneration in rat basal forebrain

    Chemico-Biological Interactions, Vol. 375

  3. Neurodegenerative Proteinopathies Induced by Environmental Pollutants: Heat Shock Proteins and Proteasome as Promising Therapeutic Tools

    Pharmaceutics, Vol. 15, Núm. 8

2022

  1. Cadmium-induced neurotoxic effects on rat basal forebrain cholinergic system through thyroid hormones disruption

    Environmental Toxicology and Pharmacology, Vol. 90

  2. Insulin Signaling Disruption and INF-γUpregulation Induce Aβ1-42and Hyperphosphorylated-Tau Proteins Synthesis and Cell Death after Paraquat Treatment of Primary Hippocampal Cells

    Chemical Research in Toxicology, Vol. 35, Núm. 12, pp. 2214-2218

  3. Single and repeated bisphenol A treatment induces ROS, Aβ and hyperphosphorylated-tau accumulation, and insulin pathways disruption, through HDAC2 and PTP1B overexpression, leading to SN56 cholinergic apoptotic cell death

    Food and Chemical Toxicology, Vol. 170

2021

  1. Bisphenol A single and repeated treatment increases HDAC2, leading to cholinergic neurotransmission dysfunction and SN56 cholinergic apoptotic cell death through AChE variants overexpression and NGF/TrkA/P75NTR signaling disruption

    Food and Chemical Toxicology, Vol. 157

2020

  1. Dysregulation of prostaglandine E2 and BDNF signaling mediated by estrogenic dysfunction induces primary hippocampal neuronal cell death after single and repeated paraquat treatment

    Food and Chemical Toxicology, Vol. 144

  2. Manganese increases Aβ and Tau protein levels through proteasome 20S and heat shock proteins 90 and 70 alteration, leading to SN56 cholinergic cell death following single and repeated treatment

    Ecotoxicology and Environmental Safety, Vol. 203

2019

  1. Manganese induced ROS and AChE variants alteration leads to SN56 basal forebrain cholinergic neuronal loss after acute and long-term treatment

    Food and Chemical Toxicology, Vol. 125, pp. 583-594

2018

  1. Cadmium alters heat shock protein pathways in SN56 cholinergic neurons, leading to Aβ and phosphorylated Tau protein generation and cell death

    Food and Chemical Toxicology, Vol. 121, pp. 297-308

  2. Cadmium induced ROS alters M1 and M3 receptors, leading to SN56 cholinergic neuronal loss, through AChE variants disruption

    Toxicology, Vol. 394, pp. 54-62

  3. SN56 neuronal cell death after 24 h and 14 days chlorpyrifos exposure through glutamate transmission dysfunction, increase of GSK-3β enzyme, β-amyloid and tau protein levels

    Toxicology, Vol. 402-403, pp. 17-27

2017

  1. Hippocampal alteration of monoaminergic neurotransmitters biosynthesis and metabolism in CNS in rats, after prenatal and postnatal exposure to chlordimeform, through sex hormones disruption

    Medicina balear, Vol. 32, Núm. 3, pp. 31-39

  2. Primary hippocampal neuronal cell death induction after acute and repeated paraquat exposures mediated by AChE variants alteration and cholinergic and glutamatergic transmission disruption

    Toxicology, Vol. 390, pp. 88-99

  3. Striatal alteration of monoaminergic neurotransmitters systems in rats, after prenatal and postnatal exposure to chlordimeform, through testosterone and estradiol disruption

    Medicina balear, Vol. 32, Núm. 2, pp. 13-22