Publicacions en què col·labora amb Sandra Rayego Mateos (13)

2022

  1. CCN2 (Cellular Communication Network Factor 2) Deletion Alters Vascular Integrity and Function Predisposing to Aneurysm Formation

    Hypertension, Vol. 79, Núm. 3, pp. E42-E55

  2. Molecular Mechanisms of Kidney Injury and Repair

    International Journal of Molecular Sciences, Vol. 23, Núm. 3

2021

  1. Deletion of delta-like 1 homologue accelerates renal inflammation by modulating the Th17 immune response

    FASEB Journal, Vol. 35, Núm. 1

2018

  1. Connective tissue growth factor induces renal fibrosis via epidermal growth factor receptor activation

    Journal of Pathology, Vol. 244, Núm. 2, pp. 227-241

2015

  1. TGF-Beta Blockade Increases Renal Inflammation Caused by the C-Terminal Module of the CCN2

    Mediators of Inflammation, Vol. 2015

  2. TNF-related weak inducer of apoptosis (TWEAK) regulates junctional proteins in tubular epithelial cells via canonical NF-κB pathway and ERK activation

    Journal of Cellular Physiology, Vol. 230, Núm. 7, pp. 1580-1593

2014

  1. IL-17A is a novel player in dialysis-induced peritoneal damage

    Kidney International, Vol. 86, Núm. 2, pp. 303-315

  2. Integrin-linked kinase plays a key role in the regulation of angiotensin II-induced renal inflammation

    Clinical Science, Vol. 127, Núm. 1, pp. 19-31

2013

  1. Connective tissue growth factor is a new ligand of epidermal growth factor receptor

    Journal of Molecular Cell Biology, Vol. 5, Núm. 5, pp. 323-335

  2. Gremlin is a downstream profibrotic mediator of transforming growth factor-beta in cultured renal cells

    Nephron - Experimental Nephrology, Vol. 122, Núm. 1-2, pp. 62-74

  3. The C-terminal module IV of connective tissue growth factor is a novel immune modulator of the Th17 response

    Laboratory Investigation, Vol. 93, Núm. 7, pp. 812-824

2012

  1. Angiotensin II contributes to renal fibrosis independently of notch pathway activation

    PLoS ONE, Vol. 7, Núm. 7

2010

  1. Statins inhibit angiotensin II/smad pathway and related vascular fibrosis, by a TGF-β-independent process

    PLoS ONE, Vol. 5, Núm. 11