Sandra Rayego Mateos-rekin lankidetzan egindako argitalpenak (13)

2022

  1. CCN2 (Cellular Communication Network Factor 2) Deletion Alters Vascular Integrity and Function Predisposing to Aneurysm Formation

    Hypertension, Vol. 79, Núm. 3, pp. E42-E55

  2. Molecular Mechanisms of Kidney Injury and Repair

    International Journal of Molecular Sciences, Vol. 23, Núm. 3

2021

  1. Deletion of delta-like 1 homologue accelerates renal inflammation by modulating the Th17 immune response

    FASEB Journal, Vol. 35, Núm. 1

2018

  1. Connective tissue growth factor induces renal fibrosis via epidermal growth factor receptor activation

    Journal of Pathology, Vol. 244, Núm. 2, pp. 227-241

2015

  1. TGF-Beta Blockade Increases Renal Inflammation Caused by the C-Terminal Module of the CCN2

    Mediators of Inflammation, Vol. 2015

  2. TNF-related weak inducer of apoptosis (TWEAK) regulates junctional proteins in tubular epithelial cells via canonical NF-κB pathway and ERK activation

    Journal of Cellular Physiology, Vol. 230, Núm. 7, pp. 1580-1593

2014

  1. IL-17A is a novel player in dialysis-induced peritoneal damage

    Kidney International, Vol. 86, Núm. 2, pp. 303-315

  2. Integrin-linked kinase plays a key role in the regulation of angiotensin II-induced renal inflammation

    Clinical Science, Vol. 127, Núm. 1, pp. 19-31

2013

  1. Connective tissue growth factor is a new ligand of epidermal growth factor receptor

    Journal of Molecular Cell Biology, Vol. 5, Núm. 5, pp. 323-335

  2. Gremlin is a downstream profibrotic mediator of transforming growth factor-beta in cultured renal cells

    Nephron - Experimental Nephrology, Vol. 122, Núm. 1-2, pp. 62-74

  3. The C-terminal module IV of connective tissue growth factor is a novel immune modulator of the Th17 response

    Laboratory Investigation, Vol. 93, Núm. 7, pp. 812-824

2012

  1. Angiotensin II contributes to renal fibrosis independently of notch pathway activation

    PLoS ONE, Vol. 7, Núm. 7

2010

  1. Statins inhibit angiotensin II/smad pathway and related vascular fibrosis, by a TGF-β-independent process

    PLoS ONE, Vol. 5, Núm. 11